Vitamin D Receptor Resistance with Dr. Sanja Tamburic ND
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- Опубліковано 11 лют 2023
- Dr. Rade interviews Dr. Sanja Tamburic, an expert in the field of vitamin D receptor resistance, autoimmune disease and complex chronic illness. They discuss all of these topics in depth, and more!
Dr. Tamburic’s website:
naturopathiclinic.com/
List of clinicians who have trained with Dr. Coimbra to learn his Vitamin D protocol:
(scroll to the bottom of the page)
www.coimbraprotocol.com/gener...
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great convo, i learned a lot
Great topic. I have few questions on this.
So increased PTH levels with normal vit d levels are the way to diagnose vid D resistance.
What about scenarios where PTH is elevated for diferent reasons while vit is normal?
In the presence of autoimmune, why not suplement high dose vit D anyway?
Also woukd genetic VLDR mutations predispose to issue.
Lastly why would vit D cause flare in chronic patients? Could it be possible vit D lowering blood calcium increases circ oxylates thus symptoms?
Great questions, as always - video reply is forthcoming :)
I have Mast Cell Activation and Ive been taking 10.000 UI vitamin D dose daily for a year and my PTH level increased and calcium decreased, maybe a higher dose would be better.
Interesting. I'm posting a video regarding this later today or tomorrow.
Hi, could high dose of vitamin D3 work for mast cell activation as well?
Wondering as well if supplementing with K2 helps to counter potential increases in calcium levels?
Dr. T has told me it doesn't really matter. I had the same thought as you...but she's the expert!
Could it be NAFLD (non-alcoholic fatty liver disease) which make the vit D resistance?
good question. I haven't heard of this link. What mechanism of action do you think might link the two?
Orthomolecularmedicine
Wondering if a genetic variant like I have might contribute to this issue / be an indicator for trialing the protocol despite having a 'normal' lab level? Quoting from Dr Rhonda Patrick's analysis of my 23andMe data - "The variant form (T) of the rs7041 polymorphism is thought to result in a protein that binds less efficiently to vitamin D. The (G;T) genotype may increase the risk of Vit D deficiency."