New Clinical Trials on NMN, SIRT6, AKG, Multivitamins & SGLT2 Inhibitors
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- Опубліковано 11 лют 2025
- In this video, Professor Andrea Maier from National University of Singapore presents about the ongoing clinical trials aimed at optimizing human health and potentially reversing the aging process. Join us as we explore groundbreaking interventions on:
• SGLT2 Inhibitors: Can these heart medications improve outcomes in healthy individuals, not just those with diabetes?
• Alpha-Ketoglutarate: A promising supplement being tested for its potential to enhance recovery and healthspan.
• Nicotinamide Mononucleotide (NMN): How this supplement could improve functional outcomes and quality of life in middle-aged individuals.
• Multivitamins and Minerals: Are they beneficial or harmful? We break down the latest research and ongoing trials.
** Credits To : ARRD & Professor Andrea Maier **
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DISCLAIMER: The information and opinions expressed in this video are for informational purposes only and are not intended to be a substitute for professional medical advice, diagnosis, or treatment. Please consult with your doctor or other qualified health provider before start taking any supplements.
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Pet peeve. I regularly see these vids on AKG - but the presenters usually fail to distinguish the FORM of AKG. A-AKG is not the same thing as Ca-AKG - the two are definitely not interchangeable from a supplementation or targeted benefit standpoint. And I've seen some discussion there can be intravenous and liposomal forms of AKG supplementation as well. I'm most often left totally confused unless one of the slides being presented notates the form. Sure would be helpful to know which form of AKG dosing/administration they are talking about....
the difference is not relevant. "Not" in the meaning of "relevancy=0". The way AKG is bound to these ligands (calcium or arginine) is through an ionic bond. This means that the AKG molecule carries a negative charge, while the calcium or arginine molecule carries a positive charge. The opposite charges attract each other, forming a bond that holds the two molecules together. AKG is anionic (neg charge) in both compounds, and dissociates in water to the same result. Some studies even suggest that A-AKG may be more readily absorbed than CaAKG. Furthermore, Calcium never should be supplemented in its cationic free form, while arginine has an additional positive effect of nitric oxide production.
this also explains why everybody shuld supplement with argAGK, while ersearchers prefer CaAGK, due to the fact that there are no short term inerferences by the calcium.
Basic knowledge in chemistry always helps to avoid talking nonsense.
@@monnoo8221 Well I only took coursework through organic chemistry in pursuit of my physics undergrad so maybe I’m missing something deeper, but while I get the surface-level of your argument I’m not so sure I agree - and I’ll provide a corollary example. If that argument were valid, one might by extension make the same argument as it relates to the various ligands used in conjunction with magnesium supplements (i.e., aspartate, chloride, lactate, citrate, glycinate, threonate, etc.). Clearly, those magnesium ligand variations have measurable consequences in terms of how our bodies use and absorb the various magnesium supplements available in the market. Moreover, while that may be a valid argument in terms of “mechanisms”, when it comes to supplementation I believe it is generally understood that the more important measure is “measured results”, preferably measured in randomized human control trials. I’m no expert in A-AKG vs. CAAKG, but from what reading I've come across it seems to me that from the vast majority of literature I’ve seen on the topic the two variations produce two different sets of results in the populations that consume them.
Hence, my pet peeve persists for now. Maybe I'm just overthinking it, or not thinking through it enough. :)
Have there been any studies comparing ca-akg, a-akg, and o-akg?
From the suppliers of these nutritional supplements 🤣
very orthodox, and not leading to anywhere.
The main problem: focusing on isolated parameters. That is, it is a cultural problem within the resarch community. Too much reductionsm, too much positivism. Single targe RCT convey and adhere to a primitive model of causationo in complex biological systems. RCTs are appropriate for a purely phenomenologial approach, where there is n theory about mechanisms, high-level treatments, like: does the paient improve if we cut his fingers insted of his arm?
What is needed instead: full scale profiling, using 1000's of parameters. Of course, the primitive empirical approach called statistics can not handle that.
Every RCT that targets on a single factor has also measure the full profile of parameters. And by that i do not refer to methylation clocks. Those are reflecting just entropy, and are the pinnacle of stupid reductionism: not using any metabolic context, no proper theory behind.