Amazing how I can pay a ton of money for my classes, yet I alwasy revert to UA-cam as my instructor. I learned more in this video than I have thus far in my class. Thanks for the great lesson! Needed it…
Thank you. 5 year medic here and I continue to get too comfortable and forget how to accurately describe disease process to Patients. thank you for the simplify this.
I was really struggling to understand pathophysiology of HF before watching it.. YOu made it so much clear than my lecturer Thank YOU SOOOOOOOMUCH!!!!!!!!!!!!!!!!
Thank you so much for making videos like these. You don't know how much it has helped me by feeling more confident about my knowledge in nursing school. Thank you so much!
I would like to express my admiration to the neatness of your work in making this subject very easy to understand . Keep up the good work and wish you all the best .
thank you so much for explaining this. I've been trying to understand this topic in google webs but thanks to u, I can understand this easily. Thank you so much
so cool... so easy listenng and watching all your lesson videos on medicine. you are AMAZING. CONGRATULATIONS. THANKS .I WILL BE FOLLOWING YOU.for ever
right side failure is the backpressure into the venous system causing hepatosplenomegaly, ascites and oedema. Left side failure is the backpressure into the pulmonary venous system causing pulmonary oedema and reduced cardiac output.
i was cured of Heart Disease by a great herbal Doctor contact him on Instagram salami_healing_herbs check that up on Instagram. with the use of his herbal remedy.
So why is the human body so good at recognizing/compensating for it's short comings but incapable of initiating a repair/restoration process to FIX THE PROBLEM.
1:36 less time to fill is not a compromise, wether with or without reduced ejection fraction due to heart muscle damage. The heart doesn’t just beat faster, it also beats more forcefully to result in faster fill time and higher pressure. Otherwise there would be zero point in increasing heart rate. We can assume the heart rate and fill time are perfectly matched, via adjusting strength of the ventricular compression. Heart rate and increased contractility go up together, eg via adrenaline. 2:38 the practitioner narrative is always: this just happens as a compensation mechanism. But dilated cardiomyopathy has direct causes, like viral infection and alcoholism, both of which lead to heart muscle damage, which makes a lot more sense than saying this happens because the heart is trying to compensate. For list of causes see eg Dilated Cardiomyopathy, 2010, the Lancet. This weakening vicious cycle is, for all I can tell, a convenient story blaming the body. It makes no sense that growing a stronger muscle to compensate would at the same time lower pumping ability. That can only be explained by continuous insult to the heart muscle. Athletes don't get dilated cardiomyopathy, they get a stronger heart. 3:30 ditto 5:39 there are mechanisms involved first that try to compensate without involving the kidneys, the so called baroreceptor reflex increases heart rate, contractibility and vasoconstriction. These all would not result in a 'vicious cycle'. Other conditions that can cause edema are kidney failure, liver disease, inflammation, dvt, NSAIDs, COPD, PE, ace inhibitors Vasodilatory edema: A common side effect of antihypertensive therapy, 2002 Effects of nonsteroidal anti-inflammatory drug therapy on blood pressure and peripheral edema, 2002 Nonsteroidal Anti-Inflammatory Drugs and Heart Failure, 2012
So then does congestive heart failure lead to respiratory Acidosis due to lungs filling with excess fluids or Metabolic Acidosis due to renal failure? If anyone has a site to refer please let me know.
Jen Greathouse ummm that's a good question. I don't think it will be very severe because when you have APO you still breath in. but yes, I would say so. lets wait what susanneheize will say about that
So does tht mean when someone experiences cardiac failure, he will experience ventricular dilation as well as oedema or dyspnea?? I mean how to know the structural changes made by the heart either dilate or hypertrophic?
sympathetic activation causing vasoconstriction which will help increasing cardiac output as compensatory mechanism. that's what I thought. wont vasodilation cause a drop in cardiac output and then reduce preload?? get back to me soon susannaheinze
Amazing how I can pay a ton of money for my classes, yet I alwasy revert to UA-cam as my instructor. I learned more in this video than I have thus far in my class. Thanks for the great lesson! Needed it…
jennifer rodriguez me too!! I feel like I should pay some youtubers instead of my own teachers who are not very helpful :(
Or we should eliminate "money" all along and do what we love because we love to understand & thus do it. 😊🤗
Agreed! Can't argue with that!
Fax
Thank you so much!! Don't stop making these videos, you have the gift of teaching. Im a nursing student and these have helped me so much.
Thank you. 5 year medic here and I continue to get too comfortable and forget how to accurately describe disease process to Patients. thank you for the simplify this.
I was really struggling to understand pathophysiology of HF before watching it.. YOu made it so much clear than my lecturer Thank YOU SOOOOOOOMUCH!!!!!!!!!!!!!!!!
Thank you so much for making videos like these. You don't know how much it has helped me by feeling more confident about my knowledge in nursing school. Thank you so much!
This was so much more helpful than my lecture. Thank you so much for breaking it down in a way that makes sense!
Thank you for making this video. Simplified but has all the essential elements. Another cramming med student is saved thank to you!
It's amazing how simple you made this concept. You are very gifted. God bless you.
I'm a nursing student, your explanation is very clear. Thank you so much!
I love your videos, you are great teacher. Excellent way to help us to understand Pathophysiology that is not the easiest one. Blessings to you.
I would like to express my admiration to the neatness of your work in making this subject very easy to understand . Keep up the good work and wish you all the best .
1st video watched on this channel and it's just perfect! such a simple and effective way of explanation/ teaching. Thoroughly enjoyed the video!
How calmly u have taught the topic that made it so easy to understand,thumps up to you❤️
thank you so much for explaining this. I've been trying to understand this topic in google webs but thanks to u, I can understand this easily. Thank you so much
WOW YOU'RE SO GOOD all the respect i really appreciate your great videos you're so smart you made the subject from complicated to simple
I understood it much better than I did in class! Thanks for the great teaching =)
Sara Temkit are you a medical student ?
That was so well explained with extra work done at home . many thanks Sussana
Fantastically described. You have a knack for teaching
Thank you for making this so understandable.
loved loved this! thank you so muchhh, have a big test on Monday, and now I'm confident I've got this... 🙂
This explanation was very very fair for a medical student even those who are not
perfect ... thank you .. saved me so much time going over my boring leture
Such a great video. I'm a nursing student and this was incredibly helpful!
Short but super effective...thankyou so much
Thank you so much, this is exactly what ive been looking for much appreciated!
You made the process very simple to understand. Thank you very much!
THANK YOU, YOU SAVED ME ❤️
Dr suassanhenze excellent demonstration and explanation on CHF thanks and regards 11/01/2020
Really i understood very well because your teaching way is very clear and satisfy able. Thanks
My Mommie had congestive heart disease. Thanks for the explanation. She was having a hard time towards the end of her life 💔 😪
so cool... so easy listenng and watching all your lesson videos on medicine. you are AMAZING. CONGRATULATIONS. THANKS .I WILL BE FOLLOWING YOU.for ever
Thank you so much
Very clear explanation - short, concise and accurate. Thanks
its the simplest explanation of CHF pathophysiology in UA-cam.
Thank you so much for making this information so easy to understand!
This is a clear and simple explanation, thank you!
Thank you! This was a fun page of notes to prepare and teach.
Thanks this was very helpful, can do please do one for myocardial infarction
U made it so intrstng ❤❤❤thanks for making such videos ❤❤
brilliantly simplified
Thanks dr for your this esay to follow feedback on congestive heart failure proud of your awareness drive 17/04/2021
Thank you so much for this upload. Helped me so much.
God Bless you, your videos are amazing.
right side failure is the backpressure into the venous system causing hepatosplenomegaly, ascites and oedema.
Left side failure is the backpressure into the pulmonary venous system causing pulmonary oedema and reduced cardiac output.
Thank you very much! for creating this, I understand it lot more.
GREAT EXPLAINATION.PLZ DO MORE VIDEOS.
Thanks for a wonderful and clear explanation.
very very beautiful and detailed.. keep it up maam.
Thank you soo much ......
This video was very very helpful😭......
God bless you❤
Your videos are gold💛
Ur video was so helpful. Pls can I get the complete chart
Really helped me grasp the compensation cycle...thanks!
all videos is very helpful in education..thanku...thanku
Thank you, you save my exam!! could'nt be more clear
simplified brilliantly
Loved ur content...🧡 from 🇮🇳
Thank you for the video! You explained it so clearly!
Thank you so so so so much!
Thanks for your video! Can you make more pathophysiology for other diseases please? Respiratory failure, afib, hepatic encephalopathy please!!!
i was cured of Heart Disease by a great herbal Doctor contact him on Instagram salami_healing_herbs check that up on Instagram. with the use of his herbal remedy.
Very easy to follow, Thank you!
Thank u very much for these clear presentation
it's excellent! it helps me a lot! thank you so much!
This was an amazing video, very helpful thank you!
Superb for basics!👌
Awesome explanation!
So why is the human body so good at recognizing/compensating for it's short comings but incapable of initiating a repair/restoration process to FIX THE PROBLEM.
this was so helpful
Thank you
This is really great
Good explanation
Thank you so much!!!
when u increase heart rate . you don't affect the diastolic phase but only the systolic phase.
Very informative 👏
in hypertrophic cardiomyopathy the sv increase but usually it's ur cardiac output that drops.
absolutely fantastic!!!!!! thankyou
Greatttt.... Thanku soo much
In heart failure hypertrophy or dilated cardiomyopathy will occur? I am confused
1:36 less time to fill is not a compromise, wether with or without reduced ejection fraction due to heart muscle damage.
The heart doesn’t just beat faster, it also beats more forcefully to result in faster fill time and higher pressure. Otherwise there would be zero point in increasing heart rate. We can assume the heart rate and fill time are perfectly matched, via adjusting strength of the ventricular compression. Heart rate and increased contractility go up together, eg via adrenaline.
2:38 the practitioner narrative is always: this just happens as a compensation mechanism. But dilated cardiomyopathy has direct causes, like viral infection and alcoholism, both of which lead to heart muscle damage, which makes a lot more sense than saying this happens because the heart is trying to compensate.
For list of causes see eg Dilated Cardiomyopathy, 2010, the Lancet.
This weakening vicious cycle is, for all I can tell, a convenient story blaming the body. It makes no sense that growing a stronger muscle to compensate would at the same time lower pumping ability. That can only be explained by continuous insult to the heart muscle. Athletes don't get dilated cardiomyopathy, they get a stronger heart.
3:30 ditto
5:39 there are mechanisms involved first that try to compensate without involving the kidneys, the so called baroreceptor reflex increases heart rate, contractibility and vasoconstriction. These all would not result in a 'vicious cycle'.
Other conditions that can cause edema are kidney failure, liver disease, inflammation, dvt, NSAIDs, COPD, PE, ace inhibitors
Vasodilatory edema: A common side effect of antihypertensive therapy, 2002
Effects of nonsteroidal anti-inflammatory drug therapy on blood pressure and peripheral edema, 2002
Nonsteroidal Anti-Inflammatory Drugs and Heart Failure, 2012
Superb ❤❤❤
THANKK YOUU MAAM ❤
simply superb
You absolutely rock!!!!!!!!!!!!!
I loved this
Brilliant!
can you please make some video about renin - angiotensin - aldosteron ?
Rinaldi Ardiansyah search interactive biology here on youtube. there are more videos that can help
Thank you!
How can I get this drawing so I can print it out and study it?
Stop the video and take its screen shot dear....😊😊
THANKS YOU ARE GREAT
Thank you
Hi! can you make videos about cardiopulmonary disorders :) thanks!
THANK YOU SOO MUCH!
HELPED me so much with my patho for my careplan =D
please share the drawing, thx much
So then does congestive heart failure lead to respiratory Acidosis due to lungs filling with excess fluids or Metabolic Acidosis due to renal failure? If anyone has a site to refer please let me know.
Jen Greathouse ummm that's a good question. I don't think it will be very severe because when you have APO you still breath in. but yes, I would say so. lets wait what susanneheize will say about that
So does tht mean when someone experiences cardiac failure, he will experience ventricular dilation as well as oedema or dyspnea?? I mean how to know the structural changes made by the heart either dilate or hypertrophic?
Via echocardiogram
sympathetic activation causing vasoconstriction which will help increasing cardiac output as compensatory mechanism. that's what I thought.
wont vasodilation cause a drop in cardiac output and then reduce preload??
get back to me soon susannaheinze
thankyou, thankyou, thankyou! you are god.
Loved it 😘😘😘😘
Chai i love love this!
wonderful
you're amazing
thaaaaanks a lot really helpful
thx