Congestive Heart Failure | Clinical Medicine
Вставка
- Опубліковано 28 тра 2024
- Premium Member Resources: www.ninjanerd.org/lectures/he...
Ninja Nerds!
In this lecture, Professor Zach Murphy will present on Congestive Heart Failure (CHF). The lecture will cover the causes and pathophysiology of CHF, including left and right heart failure and systolic, diastolic, and high-output heart failure. Clinical features and complications associated with CHF will also be discussed. A digital presentation will follow, covering the diagnosis of CHF using diagnostic imaging like chest X-rays, echocardiograms, and heart catheterization. Finally, the lecture will cover CHF treatment, including pharmacological interventions and device therapy. Enjoy the lecture and show support below!
Table of Contents:
0:00 Lab
0:07 Congestive Heart Failure (CHF) Introduction
0:50 Pathophysiology | Left Heart Failure
18:18 Pathophysiology | Right and High Output Heart Failure
28:05 Left Heart Failure Complications | Pulmonary Congestion
33:59 Left Heart Failure Complications | Cardiogenic Shock
39:49 Complications from Right Heart Failure
46:01 Diagnostic Approach
48:34 Treatment
58:00 Comment, Like, SUBSCRIBE!
Retired Ninja Nerd Lectures:
• Pathophysiology of Con...
• Treatment of Congestiv...
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#ninjanerd #cardiovascular #heartfailure
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I'm just a little nursing school kid, but I love learning the pathology. It helps me understand the concept vs memorizing it. Thank you for these digestible breakdowns!
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i started my clinical rotation before two months with surgery ward and boom i saw ninja nerds starting to focus on clinical lectures now i am more then lucky ,thank u ninja nerd team i really appreciate your work
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loads of love and respect my guy!
your vids are amazing, u covered everything i need to know for my internal med, thank you
Thank you so much for these amazing videos! Literally makes me understand medicine a whole lot better!
Just found you today. Your presentation helped me understand the process that tortured and killed my Mother and is now affecting me.
I’ve subscribed.
Me: Scarfing down my cardiac meds 😢😊
my lovely teacher!!❤
Amazing video. Thank you!
I am doing my internal medicine rotations right now, WONDERFUL, Thank you
For real
@@sherylbrenda9887 Doctors strike apparently
As usual, great job... much appreciated ❤❤❤
I like your energy on that topic!
Your enthusiasm makes my heart smile❤😊!! Thank you, Zack, from the bottom of my ❤. Your work is very important, remember that. You spread love and joy into this world of ours.
Be well💞. Take care🙏. Have faith⚓😚.
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very good lecture thank you my lovely teacher
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Maybe if I watch this a few dozen times, it will sink in. My EF was 25, still 25, 40 then 45 with meds and on a recumbent bike. Hope the Echo in two week is higher yet. And stuck with a LBBB.
Wish I could tell how much I love you
Thanks. More fun and easy than uni lectures
great video!
Thank you so much sir
Amazing
That was excellent❤️
well done sir
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great
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Thx u soo much ❤
oh my god you are the father of sceince i love you a lot keep it up
Bhaichara on top! 🎉💪🏼👍
Thank you from ❤ chambers
nice
Hey ninja nerd love the videos, probably something you might want to consider is that these videos that you privatised and then made public again, aren't classed as new videos rather from 2 days ago, so it may not pop into the subscription page of your subscribers
Yes he already explained in a video of how and where to access these videos... He put A video in the live section of his channel explaining the whole thing
As always ❤ you ninja
You guys should base your treatments sections on updated international guidelines
just wow
teach us some pharmacotherapy(applied therapeutics, dipiro)
Ventricular interdependence may exist due to growth of the left ventricle?
i love you ninja nerd
If FILLING is less then left ventricular ejection fraction should also be lowered isn't it
Wow thanks it’s very helpful. I have a few questions . Say someone is presenting with HF had a previous MI, coronary artery disease (atherosclerosis). Symptoms of tachycardia, tachypnea, high blood pressure, severely high creatinine levels, low eGFR, low haemoglobin levels, dyslipidaemia, tired upon slighted exertion, frequent cough and frothy sputum. What type of HF is that, with what ventricular (diastolic or systolic dysfunction) Or can it be both dysfunctions of the same ventricle? And based on these blood test which other organ is failing due to HF?
We give beta agonist like dobutamime to increase the contractility of the heart in case of CHF, but we give beta blockers to stop the release of renin from JG cells. Don’t these two contraindicate each other? Please explain
Contradict*
Aren’t the guidelines updated and the first line therapy is ARNI now?
What about low output heart failure and svO2 why is it high?
This awesome but how do you download notes am failing
we ❤ u
Ninja nerd ❤
Can we change Lasix with Samsca?
love you sir you are one of goats of eart ❤
Anybody know why my heart throws a fit about 20 mins into laying on my stomach? Am I never aloud to take a nap ever again? 😢 legs tingle, harder to breathe, head feels weird, really tired….
Can someone explain what is the difference between “dilated “ and “hypertrophied”.
Dilation is the expansion of the ventricle. It's like when you overfill a balloon, the walls of the balloon are now thinned out and tensed. The heart is the same way. The heart is overfilled with blood and stretches or 'dilates'.
Hypertrophy refers to increase in the size of each cardiac muscle cell, which makes them occupy more space and reduces the size of the chamber space.
@@hillithwatts7050 thank you
I'm having one question
In heart failure there is already decreased cardiac output so why are we giving drugs that decrease cardiac output?
hello ^-^ .. from what I understood: in CHF the heart is kinda of sick due whatever the cause (DM,HTN...) .. and we know as you said there is a decrease in cardiac output therefore the body does some compensation mechanisms in order to increase the cardiac output and blood pressure (like stimulating the SNS to increase heart rate and myocardial contractility through beta receptors, constriction of vessels, activation of renin-angiotensin-aldosterone-ADH system) but all these mechanisms put such a burden on the already weak heart and even weakening it more (on the long term) so we give drugs to appose these mechanisms such as beta blockers and ACE inhibitors .. hope you get the idea and if you're still confused you can go back to the retired lecture of CHF (specifically in 00:55 min he started explaining this idea).
That's the great paradox of heart failure. HF is usually caused by the body's responses to compensate for decreased output, which provide relief in the short term but worsen the problem in the long term.
I think my brother may
have AAA. The pain and swelling in his lower abdomen comes and goes. He went to the ER one day because of it.
Had a CT scan, ...doctor told him he had AAA. Next step is endoscopy. Told him to eat oatmeal and no fat . He smokes and has always eaten fried food. He has also had a quadruple bypass. He recently changed his diet and lost 40 lbs.
Doctor told him to eat oatmeal and low fat. I'm very concerned, please help.
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I am getting very stressed out watching this video
Why?
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I'm having one question
In heart failure there is already decreased cardiac output so why are we giving drugs that decrease cardiac output?
Preventing further mutation of heart shape and condition
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