Welldone ma RAAS: is a well known regulator of blood pressure and determinant of target-organ damage. It controls fluid and electrolyte balance through coordinated effects on the heart, blood vessels, and Kidneys.
Thank you for the lecture ma..it’s well explanatory. The RAAS system consists of the Renin,Angiotensin,Aldosterone system. This is activated when there is low blood volume which may occur due to haemorrhage,postpartum haemorrhage and other conditions. Then the sympathetic nervous system stimulated the kidneys in the Juxta glomerrula cells to secrets RENIN. This activated Angiotensinogen in the liver,which produces the Angiotensin 1..then the ACE comes in which is the Angiotensin converting enzyme which will convert the angiotensin 1 to 2.this is found in the lungs,kidneys and the endothelium. Immediately after the conversion,The kidney,adrenal glands and the pituitary gland are been worked on. On the kidney,it retains sodium and water,on the adrenal cortex,it also retain sodium and water and decrease potassium,and on the pituitary glands it activates the anti diuretic hormone,the vasopressin..all this is just to retain water and sodium to increase blood pressure. Now,the the alpha blockers will inhibit that ACE.so that it won’t increase the blood pressure,and there won’t be vaso. constriction..without the ACE..blood pressure won’t be increased. Adegbite Damilola Tolulope Matric no: 2023000693
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RAAS is a vital physiological pathway that regulates blood pressure, electrolyte balance, and fluid status in the body. Key Components of RAAS 1.Renin An enzyme released by the kidneys in response to decreased blood pressure or volume. 2. Angiotensinogen:A protein produced by the liver that is converted to angiotensin I by renin. 3. Angiotensin-Converting Enzyme (ACE): An enzyme that converts angiotensin I to angiotensin II. 4. Angiotensin II: A potent vasoconstrictor that increases blood pressure and stimulates the release of aldosterone. 5. Aldosterone: A hormone produced by the adrenal glands that regulates electrolyte balance and fluid status. RAAS Pathway: 1. Renin release 2. Angiotensinogen conversion to angiotensin I 3. ACE converts angiotensin I to angiotensin II 4. Angiotensin II increases blood pressure and stimulates aldosterone release 5. Aldosterone regulates electrolyte balance and fluid status
Renin angiotesin system:It is internal hormonal interaction devised by the body system to maintain adequate blood pressure in case of blood loss or shock. Organs involved are kidneys and adrenal cortex
RAAs mechanism- when the blood volume drops or blood flow to the kidney reduces, kidney produce an enzyme renin to the blood streams which help to covert plasma proteins angiotensinogen to angiotensin 1 and ACE convert angiotensin 1 to angiotensin 11 which is a potent vasoconstriction thereby increasing the blood pressure
The Renin-Angiotensin-Aldosterone System (RAAS) is a hormonal cascade critical for maintaining blood pressure, fluid, and electrolyte balance, as well as systemic vascular resistance. This system is activated in response to various physiological triggers, including low blood pressure, decreased sodium levels, or increased sympathetic nervous system activity. RAAS components are, Renin, Aldosterone, Angiotensin I, Angiotensin II etc.
RAAS mechanism- when the blood volume flow to the kidney reduces or when it drops, kidney produce an enzyme renin to the bloodstream which helps to convert plasma proteins angiotensinogen to angiotensin 1 and ACE convert angiotensin 1 to angiotensin 11 which is a potent vasoconstriction thereby increasing the blood pressure
RAAS mechanisms: when the blood volume drops or blood flow to the kidney reduces, kidney produce an enzyme renin to the blood streams which help to convert to the blood streams which help to convert plasma protein angiotensinogen to angiotensin 1 and ACE convert angiotensin 1 to angiotensin 11 which is a potent vasoconstriction there by increasing the blood pressure.
The Renin-Angiotensin-Aldosterone System (RAAS) is a hormonal cascade critical for maintaining blood pressure, fluid, and electrolyte balance, as well as systemic vascular resistance It consists of Renin, Angiotensinogen, Angiotensin I, Angiotensin converting enzyme, Angiotensin II, Aldosterone and Anti diuretic
Have you been struggling with NCLEX be it LPN, RN, or CNA, have you taken the exam for more than once? Are you taking it for the first time? Do you have a too busy schedule to study? Get to Dr Needham for your NCLEX assistance. I’m so happy I finally passed my exams thanks to him. He’s got a magical qbank which will help you become a RN
RAAS mechnics:renin is released by kidneys in response to decreased blood volume or low blood pressure which converts angiotensin 1 is converted to angiotensin ll by ACE in the lungs, angiotensin ll causes blood vessels to constrict increasing blood pressure.
When blood volume is low as a result of haemorrhage this makes the blood pressure to falls, the Juxta glomerular cells of the kidneys release an enzyme renin into the bloodstream. As renin moves in the bloodstream as it gets to the liver it then reacts with an inactive enzyme angiotensinogen which is protein produce by the liver and convert it Angiotensin I by Angiotensin converting enzyme. Angiotensin I, which is inactive (doesn’t cause any effects), flows through the bloodstream and is split into pieces by angiotensin-converting enzyme (ACE) in the lungs and kidneys and convert it to angiotensin II, an active hormone. Angiotensin II causes the muscular walls of small arteries (arterioles) to constrict (narrow), which increases blood pressure. Angiotensin II also triggers the adrenal glands to release aldosterone and the pituitary gland to release antidiuretic hormone (ADH, or vasopressin). Together, aldosterone and ADH cause your kidneys to retain sodium. Aldosterone also causes the kidneys to release (excrete) potassium through the urine. The increase in sodium in the bloodstream causes water retention. This increases blood volume and blood pressure, thus completing the renin-angiotensin-aldosterone system. SULEIMAN YAKUB 2023000962 NURSING 300L
Laven Dorcas Patrick 2023000216 Nursing department 300 Level RAAS is a hormone system that regulate blood pressure, electrolyte balance and systemic vascular resistance
Thank you so much for this wonderful lecture ma. It is well understood and please do a video on EKG. Balogun Mariam Adeola 2023000582 300L Nursing departments
Concept of RAAS The renin-angiotensin system (RAS) is one of the major control systems for blood pressure and fluid balance. The major biologically active hormone generated by this system, angiotensin (Ang) II, is produced by sequential cleavage of peptides derived from the substrate molecule angiotensinogen (Agt). The renin angiotensin aldosterone system is the system of hormones,protein, enzymes and reactions that regulate your blood pressure and blood volume on a long term basis. The major biological active hormone generated by the system, angiotensin (Ang)ii is produced by sequential cleavage of peptides derived from the substrate molecule angiotensinogen (Agt) PROCESS when there is reduction in blood flow, juxtaglomerular cells in the afferent arteriole will be stimulated and produce enzyme renin, Renin will act with Alpha globulin in blood to form angiotensin I Angiotensin I will act with unknown enzyme in the lungs to form angiotensin Il (vasoconstrictor) angiotensin Il will stimulate the adrenal cortex to produce aldosterone Aldosterone will act on the kidney tubule to increase its permeability to sodium salt leading to increase in osmotic pressure of the blood then water balance will start because they work hand in hand. By Ahmed Suleiman Ajape 2023000674 Nursing department 300L
Thank you very much ma.
I love your mode of teaching.
Welldone ma
RAAS: is a well known regulator of blood pressure and determinant of target-organ damage.
It controls fluid and electrolyte balance through coordinated effects on the heart, blood vessels, and Kidneys.
Nice one and very clear.
Thank you so very much ma
Thank you ma for this lecture
Thank you for the lecture.
This is a beautiful revision for me.
@@OluwatoyinAkindun Smiles 😍
Well understood
Thank you for the lecture ma
Very wonderful of you ma
Thank you so much ma
Nice lecture
Well understood, thanks for the lecture,ma
Interesting lecture, God bless you ma
Thank you ma, May Almighty God increase your knowledge.
@@SalaudeenKudirah Amen
Well done ma'am
Thank you ma.
Omoniyi Oluwasewa Omokorede
Well understood
Well done ma
Thank you for the lecture ma..it’s well explanatory.
The RAAS system consists of the Renin,Angiotensin,Aldosterone system.
This is activated when there is low blood volume which may occur due to haemorrhage,postpartum haemorrhage and other conditions.
Then the sympathetic nervous system stimulated the kidneys in the Juxta glomerrula cells to secrets RENIN.
This activated Angiotensinogen in the liver,which produces the Angiotensin 1..then the ACE comes in which is the Angiotensin converting enzyme which will convert the angiotensin 1 to 2.this is found in the lungs,kidneys and the endothelium.
Immediately after the conversion,The kidney,adrenal glands and the pituitary gland are been worked on.
On the kidney,it retains sodium and water,on the adrenal cortex,it also retain sodium and water and decrease potassium,and on the pituitary glands it activates the anti diuretic hormone,the vasopressin..all this is just to retain water and sodium to increase blood pressure.
Now,the the alpha blockers will inhibit that ACE.so that it won’t increase the blood pressure,and there won’t be vaso. constriction..without the ACE..blood pressure won’t be increased.
Adegbite Damilola Tolulope
Matric no: 2023000693
Have you been struggling with NCLEX be it LPN, RN, or CNA, have you taken the exam for more than once? Are you taking it for the first time? Do you have a too busy schedule to study? Get to Dr Needham for your NCLEX assistance. I’m so happy I finally passed my exams thanks to him. He’s got a magical qbank which will help you become a RN
Weldon ma, very clear
@@KudiratOluwatoyin-ll3tj Am glad
Great lecture ma
@@BinukonuRamatBola Thank you
Interesting lecture ma,kindly throw more light onEKG
Well-done ma...
Adebayo Titilayo Gift
300L
Nursing department
RAAS is a vital physiological pathway that regulates blood pressure, electrolyte balance, and fluid status in the body.
Key Components of RAAS
1.Renin An enzyme released by the kidneys in response to decreased blood pressure or volume.
2. Angiotensinogen:A protein produced by the liver that is converted to angiotensin I by renin.
3. Angiotensin-Converting Enzyme (ACE): An enzyme that converts angiotensin I to angiotensin II.
4. Angiotensin II: A potent vasoconstrictor that increases blood pressure and stimulates the release of aldosterone.
5. Aldosterone: A hormone produced by the adrenal glands that regulates electrolyte balance and fluid status.
RAAS Pathway:
1. Renin release
2. Angiotensinogen conversion to angiotensin I
3. ACE converts angiotensin I to angiotensin II
4. Angiotensin II increases blood pressure and stimulates aldosterone release
5. Aldosterone regulates electrolyte balance and fluid status
God bless you ma
Yes ma.we need the lecture on EKG
The Renin-Angiotensin Aldosterone system (RAAS)is hormonal cascade critical for maintaining blood pressure, fluid and electrolyte balance
Weldone ma
Weldon ma
Good work ma, kindly, review the EKG , Thanks 🙏
Thanks ma
Renin angiotesin system:It is internal hormonal interaction devised by the body system to maintain adequate blood pressure in case of blood loss or shock. Organs involved are kidneys and adrenal cortex
RAAs mechanism- when the blood volume drops or blood flow to the kidney reduces, kidney produce an enzyme renin to the blood streams which help to covert plasma proteins angiotensinogen to angiotensin 1 and ACE convert angiotensin 1 to angiotensin 11 which is a potent vasoconstriction thereby increasing the blood pressure
The Renin-Angiotensin-Aldosterone System (RAAS) is a hormonal cascade critical for maintaining blood pressure, fluid, and electrolyte balance, as well as systemic vascular resistance. This system is activated in response to various physiological triggers, including low blood pressure, decreased sodium levels, or increased sympathetic nervous system activity.
RAAS components are, Renin, Aldosterone, Angiotensin I, Angiotensin II etc.
Thank you very much for the lecture ma.
Please ma, we will like you to take us on EKG ma.
Will like you to also treat EKG
Well done ma
Thank you, Ma
Owolabi Olaide Lydia
300L
Nursing Science
RAAS mechanism- when the blood volume flow to the kidney reduces or when it drops, kidney produce an enzyme renin to the bloodstream which helps to convert plasma proteins angiotensinogen to angiotensin 1 and ACE convert angiotensin 1 to angiotensin 11 which is a potent vasoconstriction thereby increasing the blood pressure
RAAS mechanisms: when the blood volume drops or blood flow to the kidney reduces, kidney produce an enzyme renin to the blood streams which help to convert to the blood streams which help to convert plasma protein angiotensinogen to angiotensin 1 and ACE convert angiotensin 1 to angiotensin 11 which is a potent vasoconstriction there by increasing the blood pressure.
The Renin-Angiotensin-Aldosterone System (RAAS) is a hormonal cascade critical for maintaining blood pressure, fluid, and electrolyte balance, as well as systemic vascular resistance
It consists of Renin, Angiotensinogen, Angiotensin I, Angiotensin converting enzyme, Angiotensin II, Aldosterone and Anti diuretic
Have you been struggling with NCLEX be it LPN, RN, or CNA, have you taken the exam for more than once? Are you taking it for the first time? Do you have a too busy schedule to study? Get to Dr Needham for your NCLEX assistance. I’m so happy I finally passed my exams thanks to him. He’s got a magical qbank which will help you become a RN
Please discuss about EKG
RAAS mechnics:renin is released by kidneys in response to decreased blood volume or low blood pressure which converts angiotensin 1 is converted to angiotensin ll by ACE in the lungs, angiotensin ll causes blood vessels to constrict increasing blood pressure.
Matthew Deborah Olorunsomo
Nursing department
300L
When blood volume is low as a result of haemorrhage this makes the blood pressure to falls, the Juxta glomerular cells of the kidneys release an enzyme renin into the bloodstream.
As renin moves in the bloodstream as it gets to the liver it then reacts with an inactive enzyme angiotensinogen which is protein produce by the liver and convert it Angiotensin I by Angiotensin converting enzyme. Angiotensin I, which is inactive (doesn’t cause any effects), flows through the bloodstream and is split into pieces by angiotensin-converting enzyme (ACE) in the lungs and kidneys and convert it to angiotensin II, an active hormone.
Angiotensin II causes the muscular walls of small arteries (arterioles) to constrict (narrow), which increases blood pressure. Angiotensin II also triggers the adrenal glands to release aldosterone and the pituitary gland to release antidiuretic hormone (ADH, or vasopressin).
Together, aldosterone and ADH cause your kidneys to retain sodium. Aldosterone also causes the kidneys to release (excrete) potassium through the urine.
The increase in sodium in the bloodstream causes water retention. This increases blood volume and blood pressure, thus completing the renin-angiotensin-aldosterone system.
SULEIMAN YAKUB
2023000962
NURSING
300L
Thank you ma for the work well-done,
Firstly ma,how are submitting the assignment on cardiac medication
Secondly,I will like you to take us on EKG.
Laven Dorcas Patrick
2023000216
Nursing department
300 Level
RAAS is a hormone system that regulate blood pressure, electrolyte balance and systemic vascular resistance
Welldone ma.
Ishola Salome Mopelola
2023000074
Nursing department 300l
Well done ma
This well understood
Abegunde Adeboyewa Tolani
2023000578
300L ( Nursing Dept)
Thank you for the lecture ma
Shaibu Ojoagefu Elizabeth
2023001121
300l nursing Dept
Well done ma
Oladele Esther Omonike
2023000881
300L
Nursing Department
Obiekwe Jacinta Ifeoma
2023000775
300L Nursing department
Thank you Ma
Thank you so much ma for the lecture
Oni-Emmanuel Titilayo Taiwo
2023001071
300L
Nursing Science
Thank you ma
Esther James
2023000493
3L, Nursing Department
Abraham Elizabeth Adenike
2023001094
300L
Nursing department
Thk you for the lecture ma.
Oladele Abidemi
2023001682
300L
Nursing science
Thank you for the lecture ma
Olosunde Nafisat Adeola
2023000553
300l Nursing Dept
Well understood Ma
Abdullateef Dele Aliyu
2023000620
300l nursing department
OKOJIE OMONTESELE GRACE. NURSING DEPARTMENT 300L
Thanks ma
Emejuru Chiamaka Maryann
2023000629 300l
Nursing Department
Thank you ma
Ayandiran Christianah oluwakemi
2023000834
300L Nursing department
Nice lecture ma
Harris Faith Chiamaka
2023000050
Nursing department 300lv
Well done ma Aremu Rukayat Olaitan 2023000564 300L Nursing Department
Thank you ma
Aleru Bukola Esther
2023001605
300L Nursing science
Thank you so much for this wonderful lecture ma. It is well understood and please do a video on EKG.
Balogun Mariam Adeola
2023000582
300L
Nursing departments
Jonathan Adeoluwa Ademide
2023000107
Nursing department
300l
We are interested in ECG lecture ma'am
Nursing science department
Weldon ma pls ma we will like you to take us in EKG pls ma
Alright,i will see to that after the semester
Alright ma thanks
Ahmed Suleiman Ajape
2023000674
Nursing department
300L
Concept of RAAS
The renin-angiotensin system (RAS) is one of the major control systems for blood pressure and fluid balance. The major biologically active hormone generated by this system, angiotensin (Ang) II, is produced by sequential cleavage of peptides derived from the substrate molecule angiotensinogen (Agt).
The renin angiotensin aldosterone system is the system of hormones,protein, enzymes and reactions that regulate your blood pressure and blood volume on a long term basis.
The major biological active hormone generated by the system, angiotensin (Ang)ii is produced by sequential cleavage of peptides derived from the substrate molecule angiotensinogen (Agt)
PROCESS
when there is reduction in blood flow, juxtaglomerular cells in the afferent arteriole will be stimulated and produce enzyme renin,
Renin will act with Alpha globulin in blood to form angiotensin I
Angiotensin I will act with unknown enzyme in the lungs to form angiotensin Il (vasoconstrictor) angiotensin Il will stimulate the adrenal cortex to produce aldosterone
Aldosterone will act on the kidney tubule to increase its permeability to sodium salt leading to increase in osmotic pressure of the blood then water balance will start because they work hand in hand.
By
Ahmed Suleiman Ajape
2023000674
Nursing department
300L
Adelakun Iyanu Olajumoke
2023000393
300L
Nursing Science
Abu Ozohu Esther
Matric 2023001116
Nursing Department
300l
Nice lecture.
Joy Toyin Idowu
2022000375
400 level Nursing Dept
Hajara Muhammad
2023000080
300L Nursing department
Hajara Salisu
202201457
300 level(nursing dept)
Thanks ma'am
Joshua Emily Samson
300L
2023001403
Nursing Department
Chukwuemeka Abraham
2023000131
300L Nursing department
Weldone ma, l can't get the pronunciation ma. U mean rasistin ma
Spell the word please. I don’t get you
Aletan Esemeje Obaja
2023000702
300l
Nursing department
300l
2023002138
Emmanuel Philomina Kikah
300L nursing department
2023001018
300L nursing department
Mbaka Jane Michael
2023000415
Nursing department
300l
2023000082,300 level Nursing department.
Thanks for the lecture ma
Aduloju Abidemi Adefunke
2023000558
Nursing Science Dept 300L
Welldone ma. Thank you very much ma for this wonderful lecture 👍👍👊
Oyeleke Rebecca Olukemi
2023000754
Nursing department
300L
Well done ma
Hafsat Adamu Garafini
2023000479
300l
Nursing department
Great lecture ma.
In attendance;
2023000031
NURSING SCIENCE
More grace ma'am
I'm ADESINA CLARA ADEBOLA,
2023000838. NURSING 300 LEVEL
Thanks so much ma.Jayeola Folake Mary. 2023002181. 300L Nursing department
Ajao Omotolani ubaydat
2023000761
Nursing department
Thanks for the lecture ma
Adegboyega Esther Olamide
2023000076
Nursing department.
300L
Thank you for this beautiful lecture ma
Victoria Hajiya Idoko
2023001400
300L
Nursing
Well done ma,
Adedokun Abiola
2023000968
300l nursing department
2023000866 nursing department 300level.thanks ma for the lecture
Well done ma
2023000255
Nursing
300 level
Thank you ma for the lecture
Sherifat Adegoke
2023001957
300 L, nursing
Ebe Rhoda Abosede
2023000616
Nursing Department
300L
Adegoke Elizabeth Abiola matric no 2023001493, thank you so much 🥰 ma for the lecture, it's very interesting
Mohammad Halima Danna
2023000569
300 level nursing department
2023001432, Adefila Lateefat ODUNOLA. Nursing department 300l
Thank you ma'am for the lecture
300l nursing department
2023001794
Obi blessing
2023002130
3l nursing science
2023001383 300L Nursing Department
2023000577
Nursing department