This is one of the very few doctors who speaks the truth on this. And he only has 1.47K subscribers. There should be many more subscribers to his channel, so this is why I subscribed..
What I would recommend to everyone with ASCVD is to: Eat mostly fruits and veggies with some optional fish (for niacin and omega 3s) Supplement with niacin (improves HDL and can help some with high Lp(a), vitamin D3 and vitamin K2 as well as CoQ10 (especially if you’re taking statin) Exercise - exercise has shown improvement in your collateral coronary arterial development, so even if there are future occlusions you’ll have some circulation to cover and deliver oxygen to your heart… One study out of Switzerland showed that training around 7 hours per week (daily sessions are broken down to 3x20 min or 2x30 min sessions) along with healthy diet led to significant improvement (over 30% improvement) in coronary collateralization… So, in sum: Eat right Supplement right Exercise up to 1 hr per day👍 Finally, if you have ASCVD it’s good to consider statin therapy as it not only reduces your LDL, but potentially Apo B, is anti -inflammatory and has been shown to regress the size of plaque by as much as 5% (and 1% decrease in plaque = 20-25% risk reduction of MACE…
Agree with your points. Any thoughts on Nattokinase supplements? Started supplements recently after seeing study, suggesting soft plaque regression, discussed by Dr. Ford Brewer Taking 8000 fu’s in divided dose. TY.
1) Can you provide the evidence for the plaque regression therapy you suggest? 2) Are there studies that have been done on the role nutrition can play in reversing and stabilising plaque? Thanks Dr.
Look up: *A new approach of statin therapy in carotid atherosclerosis: Targeting indices of plaque vulnerability on the top of lipid-lowering. A narrative review* pubmed.ncbi.nlm.nih.gov/35734817/
Numerous studies have shown that either statins alone or statins in combination with other medication can regress plaque by as much as 5%… to put this in perspective, each plaque reduction of 1% is associated with 20% risk reduction… Anti-inflammatory diets with fresh fruits and veggies are the best… some fish maybe OK too (high in niacin and omega 3s)…
Very informative! It’s interesting to know that statins stabilize plaque by calcification, which is similar to the effects of long-distance running as many marathoners have a higher rates of calcified plaque compared to sedentary people.
Dear Dr Yee, thank you for sharing your valuable expertise on coronary plaque and its possible regression with high-dose statin and lifestyle changes etc. Dear Doctor, I am from India. So far I have been free of any symptoms. I have a positive family history (mother had a stroke and died 30 years ago when I was still young), father was free of any cardiovascular symptoms and died in 2020. I am probably a carrier of the 9p21.3 region gene associated with CAD and premature CAD which is common in an Asian Indian population. Your advice will be greatly appreciated. So far I have been free of any symptoms. I have a positive family history (mother had a stroke and died 30 years ago when I was a child), father was free of any cardiovascular symptoms and died in 2020. I am probably a carrier of the 9p21.3 region gene associated with CAD and premature CAD which is common in an Asian Indian population. Your advice will be greatly appreciated. I would like to have an online 30 consultation with you I had an angiography 2 weeks ago Coronary angiography: Left main stem unobstructed. Left coronary artery: 70% stenosis of prox. RIVA, 50% stenosis of prox. RIM. 70% stenosis of LCX - Proximal. 70% stenosis of Mid.LCX Right coronary artery: 80% stenosis of dist. RCA. 70% stenosis of med RCA. Diagnoses: 1. coronary triple barrel disease. - Coronary angiography elective with the diagnosis of CHD on cardiac CT on 13.07.2023: - Sign. proximal RIVA stenosis. - RCX with long wall changes and sign. Stenoses proximal and in the middle section - RIM with intermediate stenosis proximally - RCA diffuse wall-angulated with significant. Stenoses proximal and distal - Lavographically normal LVEF. no reg. motility impulses - Cardiac CT in advance (Chur) with electrical path. Ergometry: diagnosis of CHD, Ca-score >1000 - CyRF: Art. Hypertension Assessment: The 57-year-old patient is found to have quite advanced coronary three-vessel disease for his age with significant stenoses in the proximal RIVA, RCX and RCA. Stenoses in the proximal RIVA, RCX and RCA. The vessels have diffuse wall changes, but the connecting segments for an ACBP would be relatively small. The LVEF is normal. I discussed the findings with the patient at length and told him that the findings were not suitable for percutaneous revascularization. With the patient completely symptom-free, we decided to perform a cardiac MRI with a question about the extent of ischaemia. Then we discussed ACBP vs. drug therapy. In addition to the established medication, I prescribed atorvastatin and bisoprolol.
This is one of the very few doctors who speaks the truth on this. And he only has 1.47K subscribers. There should be many more subscribers to his channel, so this is why I subscribed..
What I would recommend to everyone with ASCVD is to:
Eat mostly fruits and veggies with some optional fish (for niacin and omega 3s)
Supplement with niacin (improves HDL and can help some with high Lp(a), vitamin D3 and vitamin K2 as well as CoQ10 (especially if you’re taking statin)
Exercise - exercise has shown improvement in your collateral coronary arterial development, so even if there are future occlusions you’ll have some circulation to cover and deliver oxygen to your heart… One study out of Switzerland showed that training around 7 hours per week (daily sessions are broken down to 3x20 min or 2x30 min sessions) along with healthy diet led to significant improvement (over 30% improvement) in coronary collateralization…
So, in sum:
Eat right
Supplement right
Exercise up to 1 hr per day👍
Finally, if you have ASCVD it’s good to consider statin therapy as it not only reduces your LDL, but potentially Apo B, is anti -inflammatory and has been shown to regress the size of plaque by as much as 5% (and 1% decrease in plaque = 20-25% risk reduction of MACE…
Agree with your points. Any thoughts on Nattokinase supplements? Started supplements recently after seeing study, suggesting soft plaque regression, discussed by Dr. Ford Brewer Taking 8000 fu’s in divided dose. TY.
Not what I wanted to hear...but sometimes facts aren't pleasant and help us focus on reality. Thank you. I be looking for your videos.
1) Can you provide the evidence for the plaque regression therapy you suggest? 2) Are there studies that have been done on the role nutrition can play in reversing and stabilising plaque? Thanks Dr.
Look up: *A new approach of statin therapy in carotid atherosclerosis: Targeting indices of plaque vulnerability on the top of lipid-lowering. A narrative review*
pubmed.ncbi.nlm.nih.gov/35734817/
Numerous studies have shown that either statins alone or statins in combination with other medication can regress plaque by as much as 5%… to put this in perspective, each plaque reduction of 1% is associated with 20% risk reduction…
Anti-inflammatory diets with fresh fruits and veggies are the best… some fish maybe OK too (high in niacin and omega 3s)…
Very informative!
It’s interesting to know that statins stabilize plaque by calcification, which is similar to the effects of long-distance running as many marathoners have a higher rates of calcified plaque compared to sedentary people.
Yes, exactly
excellent video, very succinct.
Thank you.
Hi Doctor please look into the use of Atherocare to remove cholesterol and plaque buildup in the arteries.
Angiogram shows 60-90% severe calcified blockage in my LAD and 90% ostial stenosis with aneurysmatic proximal in D1. What to do with that?
Dear Dr Yee, thank you for sharing your valuable expertise on coronary plaque and its possible regression with high-dose statin and lifestyle changes etc.
Dear Doctor, I am from India. So far I have been free of any symptoms. I have a positive family history (mother had a stroke and died 30 years ago when I was still young), father was free of any cardiovascular symptoms and died in 2020.
I am probably a carrier of the 9p21.3 region gene associated with CAD and premature CAD which is common in an Asian Indian population. Your advice will be greatly appreciated. So far I have been free of any symptoms. I have a positive family history (mother had a stroke and died 30 years ago when I was a child), father was free of any cardiovascular symptoms and died in 2020.
I am probably a carrier of the 9p21.3 region gene associated with CAD and premature CAD which is common in an Asian Indian population. Your advice will be greatly appreciated. I would like to have an online 30 consultation with you
I had an angiography 2 weeks ago
Coronary angiography:
Left main stem unobstructed.
Left coronary artery:
70% stenosis of prox. RIVA, 50% stenosis of prox. RIM.
70% stenosis of LCX - Proximal. 70% stenosis of Mid.LCX
Right coronary artery:
80% stenosis of dist. RCA. 70% stenosis of med RCA.
Diagnoses:
1. coronary triple barrel disease.
- Coronary angiography elective with the diagnosis of CHD on cardiac CT on 13.07.2023:
- Sign. proximal RIVA stenosis.
- RCX with long wall changes and sign. Stenoses proximal and in the middle section
- RIM with intermediate stenosis proximally
- RCA diffuse wall-angulated with significant. Stenoses proximal and distal
- Lavographically normal LVEF. no reg. motility impulses
- Cardiac CT in advance (Chur) with electrical path. Ergometry: diagnosis of CHD, Ca-score >1000
- CyRF: Art. Hypertension
Assessment: The 57-year-old patient is found to have quite advanced coronary three-vessel disease for his age with significant stenoses in the proximal RIVA, RCX and RCA. Stenoses in the proximal RIVA, RCX and RCA. The vessels have diffuse wall changes, but the connecting segments for an ACBP would be relatively small. The LVEF is normal. I discussed the findings with the patient at length and told him that the findings were not suitable for percutaneous revascularization. With the patient completely symptom-free, we decided to perform a cardiac MRI with a question about the extent of ischaemia. Then we discussed ACBP vs. drug therapy. In addition to the established medication, I prescribed atorvastatin and bisoprolol.
Good video sir. Please continue your videos
Thank you, Appreciated.
Please post the RCT of your recommendation
What about Policosanol? Some say Policosanol remove plaques
PLEASE REMOVE THE MUSIC BACKGROUND.
THAT DISTRACTS THE AUDIENCE.