1:15 Review of myopia 2:45 Intro to Emmetropization 3:25 Growth of the Eye 6:18 Inducing Myopia 16:50 Accommodation 22:45 Control Not from Fovea 29:21 Detecting Focus 31:45 Effect of Light 36:42 Dopamine 41:50 Emmetropization 45:55 Neitz Alternate Theory
Very informative video. Summarized all the content related to emmetropization beautifully. Can't believe I learnt about all this in just an hour because it took me years to even understand a single word out of those research papers...
13:05 The lens defocus tells the eye where to focus, thus lengthening or shortening the eye in the process. The light doesn't tell the eye where to go, it solely allows the muscles of the eyes to activate, when the light is more intense the eye muscles respond more and shortens the eye. When light conditions are low, the muscles don't activate and this under-use lenhetens the eyes. A stronger light should therefor not have any particular effect in this myopic lens defocus because normal light conditions are enough for the eye muscles to maintain adaption to the lens. The more intense the light, the more the eye wants to shorten, but it cannot bypass the limitation of the focus. When the current focus is at emmetropia, the light will not further induce shortening and instead stabilize towards this limitation.
WGH first thanks G-d for your very good video , could you please tell in this video in about minute 28:00 there was an experiment that checked half blurring , by the angle , by putting half path of the light go , half blurred , but the result could not be quite understood what was it , did half eye elongate (retina grow) , and half eye did not elongate ( did not grow ) ? , or all eye elongate , could you please tell , what was the result , half eye elongate , or full eye retina elongate grow ? Why the question comes , since in minute 28:23 it is said that the y axis is the deferent between the two EYES , while it was expected to be said the deferent between the tow SIDES So could you please tell , what was the result , half eye elongate , or full eye retina elongate grow ?
Very good videos but dated. Beyond gold, there is platinum: Medina A. The cause of myopia development and progression: Theory, evidence, and treatment. Surv Ophthalmol. 2022 Mar-Apr;67(2):488-509. doi: 10.1016/j.survophthal.2021.06.005. Epub 2021 Jun 25. PMID: 34181975.
@@myopiaprogression7574 Is that Dr. Antonio Medina? E.E. Yes, correct. The outdated theory, is the Box Camera theory of J. Kepler. But times change. The fundamental eye always controls its refractive STATE, to its average of Accommodation.
@@otiebrown9999 Random question, but if somene is in their mid twentees and developes myopia, by high pressure, because I didn't go to sleep and had 24/7 upclose vision, is it possible to reverse it? I'm like -1.5 or such, with astigmatism on one eye. Or is that still not possible no matter what? I had perfect vision, and then after 3weeks I went from 20/20 to basically -1 :/ all my fault, and I know exactly why... I would even patch my eye when it used to hurt just so I can sit in front of the computer, and squint the other to relieve some pain, and did that for the 3weeks - andit took me two months to get rid of the pain although my right eye still has some pressue in after almost two years and its a bit uncomfortable
@Chaig Blackwell, Your videos is interesting but when I see pictures on video I'm confused. 7:25 on the picture I see that PLUS lens (for example +3 diopt) induces Myopia, and MINUS (for example -3 diopt) induces Hyperopia. On 7:55 You are talking about "for example a MINUS lens creates hyperopic defocus which causes the eye to lengthen resulting." Axial length is grown as I understood if focus is on sclera and grow of AL induces Myopia.
This video presents the conclusions of some investigators. The problem is that many of the papers presented here have unsupported conclusions and many have been shown to be flawed. The important papers on myopia are missing.
11:00 Dr. Craig Blackwell, At least you get this part accurately presented. Each eyes is "servoing", to its AVERAGE value of ACCOMMODATION. A plus and minus 3 lens, demonstrates this servoing response, correctly. There is no "Emmetropization", at all. I know you are a medical doctor, and not a control systems engineer. You were taught Kepler's theory, and simply do not understand why all eyes are controlling their refractive STATE, to their Average Accommodation signal.
If you said that all eyes are dynamic systems, at birth, you would be very accurate - in science. No eye was ever a "frozen cameras", as you believe it to be. That is the scientific issue we should discuss, on a level field. Prevention is possible, if you will use a plus for all closs work, before you go below 20/50, on your home Snellen.
Gene therapy for red green color blindness please. In my opinion, its visual impairment. I cant discriminate some colors. I cant get my dream professions. My sight of color is very limited. Please help me and 350 million people whom color blind🙏😭
Beautiful:
All normal eyes, control their refractive STATE , to THEIR AVERAGE VALUE OF ACCOMMODATION.
A perfect scientific truth.
1:15 Review of myopia
2:45 Intro to Emmetropization
3:25 Growth of the Eye
6:18 Inducing Myopia
16:50 Accommodation
22:45 Control Not from Fovea
29:21 Detecting Focus
31:45 Effect of Light
36:42 Dopamine
41:50 Emmetropization
45:55 Neitz Alternate Theory
This whole video supports jake steiner and the whole endmyopia theories
Very informative video. Summarized all the content related to emmetropization beautifully. Can't believe I learnt about all this in just an hour because it took me years to even understand a single word out of those research papers...
Myopia Progression
0 seconds ago
Very good videos but dated. Most content has been superseded.
Thanks for this, expecially an explanation of SightGlass.
Thank you, and good catch on video 5. Numbers were correct. Graph label error fixed and rerecorded.
13:05 The lens defocus tells the eye where to focus, thus lengthening or shortening the eye in the process. The light doesn't tell the eye where to go, it solely allows the muscles of the eyes to activate, when the light is more intense the eye muscles respond more and shortens the eye. When light conditions are low, the muscles don't activate and this under-use lenhetens the eyes.
A stronger light should therefor not have any particular effect in this myopic lens defocus because normal light conditions are enough for the eye muscles to maintain adaption to the lens. The more intense the light, the more the eye wants to shorten, but it cannot bypass the limitation of the focus. When the current focus is at emmetropia, the light will not further induce shortening and instead stabilize towards this limitation.
WGH first thanks G-d for your very good video ,
could you please tell in this video in about minute 28:00 there was an experiment that checked half blurring , by the angle , by putting half path of the light go , half blurred , but the result could not be quite understood what was it , did half eye elongate (retina grow) , and half eye did not elongate ( did not grow ) ? , or all eye elongate , could you please tell , what was the result , half eye elongate , or full eye retina elongate grow ?
Why the question comes , since in minute 28:23 it is said that the y axis is the deferent between the two EYES , while it was expected to be said the deferent between the tow SIDES
So could you please tell , what was the result , half eye elongate , or full eye retina elongate grow ?
this video is gold
Very good videos but dated. Beyond gold, there is platinum: Medina A. The cause of myopia development and progression: Theory, evidence, and treatment. Surv Ophthalmol. 2022 Mar-Apr;67(2):488-509. doi: 10.1016/j.survophthal.2021.06.005. Epub 2021 Jun 25. PMID: 34181975.
@@myopiaprogression7574
Is there a UA-cam video?
@@myopiaprogression7574 Is that Dr. Antonio Medina? E.E.
Yes, correct.
The outdated theory, is the Box Camera theory of J. Kepler.
But times change.
The fundamental eye always controls its refractive STATE, to its average of Accommodation.
@@mballer Hi Mr. Ball,
I developed some of the videos.
But they are Engineering Models, AMF difficult to understand.
@@otiebrown9999 Random question, but if somene is in their mid twentees and developes myopia, by high pressure, because I didn't go to sleep and had 24/7 upclose vision, is it possible to reverse it? I'm like -1.5 or such, with astigmatism on one eye.
Or is that still not possible no matter what? I had perfect vision, and then after 3weeks I went from 20/20 to basically -1 :/ all my fault, and I know exactly why... I would even patch my eye when it used to hurt just so I can sit in front of the computer, and squint the other to relieve some pain, and did that for the 3weeks - andit took me two months to get rid of the pain although my right eye still has some pressue in after almost two years and its a bit uncomfortable
@Chaig Blackwell, Your videos is interesting but when I see pictures on video I'm confused. 7:25 on the picture I see that PLUS lens (for example +3 diopt) induces Myopia, and MINUS (for example -3 diopt) induces Hyperopia. On 7:55 You are talking about "for example a MINUS lens creates hyperopic defocus which causes the eye to lengthen resulting." Axial length is grown as I understood if focus is on sclera and grow of AL induces Myopia.
This video presents the conclusions of some investigators. The problem is that many of the papers presented here have unsupported conclusions and many have been shown to be flawed. The important papers on myopia are missing.
Yes.
There is not one word, about Prevention, when still at 20/40.
It is that first minus that is certainly going to create stair-case myopia.
Which papers are missing?
How all normal controls their long-term refractive STATE, is indeed the $64,000 scientific question.
It is never a medical issue, nor question.
11:00
Dr. Craig Blackwell,
At least you get this part accurately presented.
Each eyes is "servoing", to its AVERAGE value of ACCOMMODATION.
A plus and minus 3 lens, demonstrates this servoing response, correctly.
There is no "Emmetropization", at all.
I know you are a medical doctor, and not a control systems engineer.
You were taught Kepler's theory, and simply do not understand why all eyes are controlling their refractive STATE, to their Average Accommodation signal.
If you said that all eyes are dynamic systems, at birth, you would be very accurate - in science.
No eye was ever a "frozen cameras", as you believe it to be.
That is the scientific issue we should discuss, on a level field.
Prevention is possible, if you will use a plus for all closs work, before you go below 20/50, on your home Snellen.
Gene therapy for red green color blindness please. In my opinion, its visual impairment. I cant discriminate some colors. I cant get my dream professions. My sight of color is very limited. Please help me and 350 million people whom color blind🙏😭