very general big picture ideas. no good if you want specifics like to learn about levo, norepi, dobutamine vs. dopamine (one very brief blip comparing some meds)
californiaMohammed thanks Mohammed. I' aiming at medical students- so I think it's best to get the big pictures first. Are you physician or student? All the bets Rob
Hi, Just a comment to feedback your video. It's nice, and explains some very basic concepts necessary to understand why we use certain medications..... but there are some things to address: 1. You DON'T explain fully the basic pharmacology of some of the medications, which was what I was looking for in the first place and why I came to your video. You just explain adrenaline and noradrenaline. But you don't explain very well Dopamine and where this D receptors are located and what the effect is. 2. You don't mention Dobutamine. (I know it's not a catecholamine, but its effects are also worth to mention) 3. You have a conceptual mistake... You say afterload is the pressure against which the LV has to contract.... And then you say that vasopressors are the drugs you use to ameliorate this? If you vasoconstrict ... wouldn't it increase the pressure against which the LV is contracting and pushing blood? I would say the way to increase afterload is to diminish the pressure in the peripheral vasculature, especially in the aorta, so the LV doesn't have to make so much effort. Anyway thank you.
very general big picture ideas. no good if you want specifics like to learn about levo, norepi, dobutamine vs. dopamine (one very brief blip comparing some meds)
californiaMohammed thanks Mohammed. I' aiming at medical students- so I think it's best to get the big pictures first. Are you physician or student?
All the bets
Rob
Hi,
Just a comment to feedback your video. It's nice, and explains some very basic concepts necessary to understand why we use certain medications..... but there are some things to address:
1. You DON'T explain fully the basic pharmacology of some of the medications, which was what I was looking for in the first place and why I came to your video. You just explain adrenaline and noradrenaline. But you don't explain very well Dopamine and where this D receptors are located and what the effect is.
2. You don't mention Dobutamine. (I know it's not a catecholamine, but its effects are also worth to mention)
3. You have a conceptual mistake... You say afterload is the pressure against which the LV has to contract.... And then you say that vasopressors are the drugs you use to ameliorate this? If you vasoconstrict ... wouldn't it increase the pressure against which the LV is contracting and pushing blood? I would say the way to increase afterload is to diminish the pressure in the peripheral vasculature, especially in the aorta, so the LV doesn't have to make so much effort.
Anyway thank you.
She does mention Dobutamine.
Increased SVR = increased afterload