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A *low-carb-high-fat diet* (LCHF) lowers our general level of body-wide *inflammation,* thereby easing or eliminating many inflammatory cpnditrons like Crohn's disease, IBS, ulcerative colitis, fibromyalgia, rheumatoid arthritis, COPD, and so on. (By “high fat” we mean *healthy* fats, not seed oils or hydrogenated oils.) A LCHF diet also fosters metabolic health, and prevents cardiovascular disease. *Don’t let doctors seduce you into taking toxic statins.* High LDL cholesterol is not a problem as long as we have low triglycerides, low inflammation, and a low LDL particle count.
Thanks for this useful insight. Could I dare to make a suggestion: stop the sliding transfer between frames, it's a distraction from the main topic - sorry!
Probably the most comprehensive explanation I’ve heard. On a statin now, so will definitely be checking for the test mentioned to confirm. Have all the side effects is stain too, so this could lead to getting off of a med I don’t need. Thanks!
Many thanks. Been keto then carnivore for 1.5 years. HDL-74; Trig-66, but LDL-246. I suspect all is good. (I feel better than ever!) My dr was concerned about the LDL, but he’s pretty reasonable. He agreed to order the advanced test.
Uhgg... all my numbers have gotten SO much better. HDL up, triglycerides way down, BP way down, awesome HDL/Try ration is excellent at 1.5. Pattern A LDL. But my lDL-c and Ldl-P have gone WAY up. Just don't know what to do.
The C in LDL- C stands for calculated. It's not a direct measurement of LDL. You can get a direct LDL which is a measurement of the LDL in the blood. But usually the calculation is close enough. Close enough is okay when you're generally looking at a snapshot in time because cholesterol levels can be affected by so many things. So definitely not accurate enough to prescribe statins. I consider the basic panel with the typical LDLC/ calculation as a screening test. When screening tests show that further testing needs to be done then a direct LDL can be ordered or the full particle work up. It's difficult because so many doctors do not have protocols on how to deal with the particle information. It's just not an up-to-date practice in conventional medicine. So we need to continue to show up to the doctor's office with these particle numbers so a truly informed conversation can be had.
Thanks for this video! I'd like to get a lipo profile to see if my 174 LDL is mostly large/buoyant or small/dense and how the two relate. I do miss my egg yolks 🍳
In my opinion , instead of testing u have big particle or small of Ldl , work on cause of injury on vessels and correct them like high blood pressure and diabetes which causes injury to vessels and ldl come to patch , cholesterol bad or small or big is waste of time . High pressure and sugar is controlled in three month with whole food plant diet . So instead of treatment go work on cause . Statin if do any good not because it lower cholesterol but it has anti inflammatory property , which can be achieved by aspirin low dose therapy or holistic approach turmeric and ginger tea.
As of yesterday I have absolute proof that the carnivore diet causes heart disease, in me anyway. I'm 57, 1.75m and 76kg, have never smoked always been relatively fit, drink in moderation and have never touched drugs of any kind including weed. I’m quite active and have a labour intensive job, lots of climbing around etc. I ride dirt bikes which is a proper workout. I started keto in 2015 and then went full carnivore in 2019. I had a STEMI in July 2021, 99% blockage in LAD, needed a bypass but we were in the third wave of COVID and the ventilators were not allowed to be used. Cardiologist was forced to stent into an aneurysm. I was put on a low dose statin 5mg Crestor. Cardiologist did another angiogram 6 weeks later to check her stent, while there she checked all other arteries, found one non flow restricting lesion in RCA, 10%, which she wasn't concerned about. I continued the carnivore diet, but was even stricter, no cheating and took my statin with blood thinners etc. On Tuesday this week I had another heart attack, 99% blockage in the RCA. Blood sugar 4.5, blood pressure 120/80 so no concerns there. Cardiologist made no bones about it that my high cholesterol was the cause, and with the evidence I have how can I argue. So to summarize, I had the gold standard test and no blockages found, 1.5 years later on strict carnivore, full blown heart attack. To say I'm not upset about this is putting it mildly.
I'm so sorry to hear this happened to you, David! Thanks for sharing your story. Everyone really is different in their nutritional needs. While I'm sure you had your own reasons for doing so, we don't advocate carnivore due to a lack of anti-inflammatory nutrients. I really hope you get better quickly and can find a dietary approach that keeps you healthy!
@@drmorgannolte I don't have these numbers, I was very surprised at high my Triglycerides were and how low my HDL was, so you can draw some inference there.
So my LDL-C being 350 and most likely rise much more at the rate I have been going with the Keto. I am more than certain. I guess I will do an NMR lipoprofile test with IR marker. I am very certain that it will show insulin resistance since I just recenly lowered the A1C recently from 5.7 to 5.3. As you said, you are insulin resistant long before anything shows up in terms of A1C. if my LDL-P is under 1000, I have the best of all world? Assuming and neglecting the IR marker. What a trip.
So what does one do if they have an extremely high ldl-p? I’ve been eating a strict animal based diet for well over a year and my ldl-p is still extremely high.
They are not well versed on LMHR studies. They have a lot invested in their old models and are resistant to new data that conflicts with their ingrained model.
Well, just last year the medical profession was calling the two types of LDL particles high density & low density. Which is LDL-P? High or low density?
Both are correct. A LDL particle can be high density, or low density. It depends on the amount of cholesterol, protein (higher density), and other substances in the lipoprotein particle.
Triglycerides and Cholesterol are Not water soluble and needs to be Transported in lipoproteins. There different Typs of This lipoproteins. So lipoproteins are the Transporter and Triglyceride and Cholesterol are the Cargo. Triglycerides is for Energy, you burn it in your cells... Cholesterol has a structural function, cellwalls Hormones etc. but not Energy. Amongs all the different lipoproteins in your system is only one, which has No risk attached. This is HDL. The only Exception is very Low or very high concentrations. The big Range in the middle is harmless. All the other lipoproteins are "Bad" VLDL, IDL,LDL, LP(a), Chylomicron remnants, Chylomicron. Only one Exception Chylomicron are to big and can be seen as less problematic, but when the get smaller below the threshold of 70nm then they become "Bad" too. In This state we call them Chylomicron remnants. Now the important part .... we Care about the number of Transporter and Not the amount of Cargo. But we measure the Cargo to make an estamation for number of Transporters. The different lipoproteins can be seperated by the Cargo they carry. LDL contains nearly exclussively Cholesterol. VLDL, IDL, contains mostly Triglycerides. So by measuring Triglyceride and Cholesterol, we can estamate roughly the number of all "Bad" Transporter. This is the reason why high Triglycerides are "Bad". They travel in different Transporters then Cholesterol, But we want less of all "BAD" Transporter and don't Care about the Cargo. We Just use the Cargo to estamate the number of Transporters. Heart disease is linked to a high number of "BAD" lipoproteins (Mostly LDL and VLDL) you want them both together as Low AS possible. VLDL carries Mostly Triglyceride. So by having high Triglyceride you probably having high VLDL numbers and having Higher risk of heart disease. The Second reason is that high Triglyceride are a Symptom of insulinresistence, prediabetes, and Diabetes. This mean This patient probably suffer under chronic elevated blood sugar Level. Chronic High Blood sugar by himself is linked to heart disease.
In the last six months, I exercised more, lost weight and cut fats and sugars/carbs ( I guess not enough though) and LDL went up a bit, HDL up a bit, Trigs down a little. First NMR was done and LDL-p and small LDL was in high range. Overall size was Type A (good) but I hear the high LDL-p # trumps that. What is best way to lower LDL-p # and small size #'s? Also, I thought LDL-P increased with Triglycerides and my Trigs went down.
If one on keto Carnivore diet, non hdl and LDL slightly a bit high and didnt fast for twelve hours before take the test. What does this mean ? If this ok ?
If statins have absolutely no effect on ldlp and they lower ldlc then is it correct to say that we are increasing the chance of atherosclerotic event by putting every poor trusting patient with high cholesterol on statin Drug
Not completely. I really liked Dr. Peter Attia's book Outlive as I thought he did an excellent job explaining when statins may be appropriate for cardiovascular disease prevention. He also did wonderfully at explaining how atherosclerosis develops. He discusses Lp(a) and apo B which isn't covered in this video.
🥳Zivli, our online course and coaching program to help you reverse insulin resistance for long-term weight loss and disease prevention is OPEN now through January 10, 2023 at 3 PM Central Time. To learn more and join today, go to www.zivli.com/join.
A *low-carb-high-fat diet* (LCHF) lowers our general level of body-wide *inflammation,* thereby easing or eliminating many inflammatory cpnditrons like Crohn's disease, IBS, ulcerative colitis, fibromyalgia, rheumatoid arthritis, COPD, and so on. (By “high fat” we mean *healthy* fats, not seed oils or hydrogenated oils.) A LCHF diet also fosters metabolic health, and prevents cardiovascular disease. *Don’t let doctors seduce you into taking toxic statins.* High LDL cholesterol is not a problem as long as we have low triglycerides, low inflammation, and a low LDL particle count.
Where is your link to Carol’s story?
Best description and graphic I’ve heard explaining this important topic. Thank you!
Glad it was helpful! This topic took me a while to be able to explain so I'm glad I did so clearly.
The particle size hypothesis is also just that a hypothesis and currently only supported by associations
Same with every other lipoprotein hypothesis, no?
It’s all a hypothesis - just depends on who’s study you trust
Thanks for this useful insight. Could I dare to make a suggestion: stop the sliding transfer between frames, it's a distraction from the main topic - sorry!
Probably the most comprehensive explanation I’ve heard. On a statin now, so will definitely be checking for the test mentioned to confirm. Have all the side effects is stain too, so this could lead to getting off of a med I don’t need. Thanks!
Great video! 👍🏻. Thanks Dr. Nolte. I’ve been low carb for over 40 years and this is definitely the way to go to be healthy. ❤️
Thanks so much for watching and leaving a positive comment, I hope you found this video valuable!
Many thanks. Been keto then carnivore for 1.5 years. HDL-74; Trig-66, but LDL-246. I suspect all is good. (I feel better than ever!) My dr was concerned about the LDL, but he’s pretty reasonable. He agreed to order the advanced test.
Uhgg... all my numbers have gotten SO much better. HDL up, triglycerides way down, BP way down, awesome HDL/Try ration is excellent at 1.5. Pattern A LDL.
But my lDL-c and Ldl-P have gone WAY up. Just don't know what to do.
The C in LDL- C stands for calculated. It's not a direct measurement of LDL.
You can get a direct LDL which is a measurement of the LDL in the blood. But usually the calculation is close enough. Close enough is okay when you're generally looking at a snapshot in time because cholesterol levels can be affected by so many things. So definitely not accurate enough to prescribe statins. I consider the basic panel with the typical LDLC/ calculation as a screening test. When screening tests show that further testing needs to be done then a direct LDL can be ordered or the full particle work up. It's difficult because so many doctors do not have protocols on how to deal with the particle information. It's just not an up-to-date practice in conventional medicine. So we need to continue to show up to the doctor's office with these particle numbers so a truly informed conversation can be had.
Thanks for watching and leaving this thoughtful comment.
Thanks for this video! I'd like to get a lipo profile to see if my 174 LDL is mostly large/buoyant or small/dense and how the two relate. I do miss my egg yolks 🍳
Glad it was helpful! That would be a good idea!
Very informative video
Glad you found it helpful!
A new paper shows the relationship between BMI and LDL is the real factor Not Fat in the diet.
In my opinion , instead of testing u have big particle or small of Ldl , work on cause of injury on vessels and correct them like high blood pressure and diabetes which causes injury to vessels and ldl come to patch , cholesterol bad or small or big is waste of time . High pressure and sugar is controlled in three month with whole food plant diet . So instead of treatment go work on cause . Statin if do any good not because it lower cholesterol but it has anti inflammatory property , which can be achieved by aspirin low dose therapy or holistic approach turmeric and ginger tea.
Plants cause high blood sugar bruh! Eating carnivore will correct your blood sugar In Literally a week! AAALLLLLL SUGAR COMES FROM PLANTS!
As of yesterday I have absolute proof that the carnivore diet causes heart disease, in me anyway.
I'm 57, 1.75m and 76kg, have never smoked always been relatively fit, drink in moderation and have never touched drugs of any kind including weed.
I’m quite active and have a labour intensive job, lots of climbing around etc. I ride dirt bikes which is a proper workout.
I started keto in 2015 and then went full carnivore in 2019.
I had a STEMI in July 2021, 99% blockage in LAD, needed a bypass but we were in the third wave of COVID and the ventilators were not allowed to be used.
Cardiologist was forced to stent into an aneurysm. I was put on a low dose statin 5mg Crestor.
Cardiologist did another angiogram 6 weeks later to check her stent, while there she checked all other arteries, found one non flow restricting lesion in RCA, 10%, which she wasn't concerned about.
I continued the carnivore diet, but was even stricter, no cheating and took my statin with blood thinners etc. On Tuesday this week I had another heart attack, 99% blockage in the RCA. Blood sugar 4.5, blood pressure 120/80 so no concerns there.
Cardiologist made no bones about it that my high cholesterol was the cause, and with the evidence I have how can I argue. So to summarize, I had the gold standard test and no blockages found, 1.5 years later on strict carnivore, full blown heart attack.
To say I'm not upset about this is putting it mildly.
Thanks for posting. My sister and I had been contemplating going carnivore. Glad I saw your post.
I'm so sorry to hear this happened to you, David! Thanks for sharing your story. Everyone really is different in their nutritional needs. While I'm sure you had your own reasons for doing so, we don't advocate carnivore due to a lack of anti-inflammatory nutrients. I really hope you get better quickly and can find a dietary approach that keeps you healthy!
I'd also be really curious about your LDL particle number and insulin level.
@@drmorgannolte I don't have these numbers, I was very surprised at high my Triglycerides were and how low my HDL was, so you can draw some inference there.
@@drmorgannolte Thank you, I can't afford to get it wrong again.
Do you have any data on whether a keto/low saturated fat diet raises LDL as much as a keto/high saturated fat diet?
Great info, do you have a reference article that I can show my doctor? She is pushing statins, based on entire LDL score
So my LDL-C being 350 and most likely rise much more at the rate I have been going with the Keto. I am more than certain.
I guess I will do an NMR lipoprofile test with IR marker. I am very certain that it will show insulin resistance since I just recenly lowered the A1C recently from 5.7 to 5.3.
As you said, you are insulin resistant long before anything shows up in terms of A1C.
if my LDL-P is under 1000, I have the best of all world? Assuming and neglecting the IR marker.
What a trip.
So what does one do if they have an extremely high ldl-p? I’ve been eating a strict animal based diet for well over a year and my ldl-p is still extremely high.
Dr Muhammad Alo, cardiologist and Dr Thomas Dayspring, world's leading lipid are wrong? Please explain.
They are not well versed on LMHR studies. They have a lot invested in their old models and are resistant to new data that conflicts with their ingrained model.
Well, just last year the medical profession was calling the two types of LDL particles high density & low density. Which is LDL-P? High or low density?
Both are correct. A LDL particle can be high density, or low density. It depends on the amount of cholesterol, protein (higher density), and other substances in the lipoprotein particle.
What about high triglycerides?
Triglycerides and Cholesterol are Not water soluble and needs to be Transported in lipoproteins. There different Typs of This lipoproteins. So lipoproteins are the Transporter and Triglyceride and Cholesterol are the Cargo. Triglycerides is for Energy, you burn it in your cells... Cholesterol has a structural function, cellwalls Hormones etc. but not Energy.
Amongs all the different lipoproteins in your system is only one, which has No risk attached. This is HDL.
The only Exception is very Low or very high concentrations. The big Range in the middle is harmless.
All the other lipoproteins are "Bad" VLDL, IDL,LDL, LP(a), Chylomicron remnants, Chylomicron. Only one Exception Chylomicron are to big and can be seen as less problematic, but when the get smaller below the threshold of 70nm then they become "Bad" too. In This state we call them Chylomicron remnants.
Now the important part .... we Care about the number of Transporter and Not the amount of Cargo. But we measure the Cargo to make an estamation for number of Transporters.
The different lipoproteins can be seperated by the Cargo they carry. LDL contains nearly exclussively Cholesterol. VLDL, IDL, contains mostly Triglycerides.
So by measuring Triglyceride and Cholesterol, we can estamate roughly the number of all "Bad" Transporter. This is the reason why high Triglycerides are "Bad". They travel in different Transporters then Cholesterol,
But we want less of all "BAD" Transporter and don't Care about the Cargo. We Just use the Cargo to estamate the number of Transporters.
Heart disease is linked to a high number of "BAD" lipoproteins (Mostly LDL and VLDL) you want them both together as Low AS possible. VLDL carries Mostly Triglyceride. So by having high Triglyceride you probably having high VLDL numbers and having Higher risk of heart disease.
The Second reason is that high Triglyceride are a Symptom of insulinresistence, prediabetes, and Diabetes. This mean This patient probably suffer under chronic elevated blood sugar Level. Chronic High Blood sugar by himself is linked to heart disease.
In the last six months, I exercised more, lost weight and cut fats and sugars/carbs ( I guess not enough though) and LDL went up a bit, HDL up a bit, Trigs down a little. First NMR was done and LDL-p and small LDL was in high range. Overall size was Type A (good) but I hear the high LDL-p # trumps that. What is best way to lower LDL-p # and small size #'s? Also, I thought LDL-P increased with Triglycerides and my Trigs went down.
Same problem...my trig -41mg/dl, small dense ldl-49mg/dl, ldl-96mg/dl
If one on keto Carnivore diet, non hdl and LDL slightly a bit high and didnt fast for twelve hours before take the test. What does this mean ? If this ok ?
If statins have absolutely no effect on ldlp and they lower ldlc then is it correct to say that we are increasing the chance of atherosclerotic event by putting every poor trusting patient with high cholesterol on statin Drug
Not completely. I really liked Dr. Peter Attia's book Outlive as I thought he did an excellent job explaining when statins may be appropriate for cardiovascular disease prevention. He also did wonderfully at explaining how atherosclerosis develops. He discusses Lp(a) and apo B which isn't covered in this video.