Webinar 2018 - Iron & RLS
Вставка
- Опубліковано 6 вер 2024
- This webinar was presented by Christopher Earley, MB, BCh, PhD, FRCPI, Professor of Neurology at Johns Hopkins School of Medicine, where he is also co-director of the Johns Hopkins Center for Restless Legs, a certified RLS Quality Care Center.
In this webinar you will learn about the history of iron, the basic understanding of iron biology, including the basis for the definition of iron deficiency, and clinical trials that have used iron treatment.
All webinar recordings can be viewed in the Member Portal of the Foundation's website at www.rls.org.
View our program and register for upcoming webinars here: www.rls.org/get-involved/education-events
Thank you so much for this! I feel like I have a much better understanding of what's going on in my body. Now I just have to convey this to my doctor.
I was a CNA and I wish I could go back in time and tell my former patients about this..
My mother has been doing yoga..no medications,no drugs..just alot of water,less fatty food,..and iron..
Is there a simple resource I can take to my doctor that reflects these treatment protocols? My doctor is hesitant to use infusions because "you can just supplement," even though I have gut issues.
I don't understand why the Dr. thinks we shouldn't test the ferritin levels of kids of RLS sufferers unless the kids are having RLS symptoms. Since he said low ferritin or I.D. Anemia can be present for years before someone has RLS symptoms, wouldn't it be better to test these kids and treat their possible deficiency, so they never have to experience RLS symptoms in the first place?
Low ferritin levels can mask iron overload, the opposite condition, so unless they are using a better test I wouldn’t test for iron.
Doctor, my two leg disorder move leg, night can not sleep,1 minit or 2 minit after automatique move leg, this problem longtime, but no pain leg, just move leg day And night, night so mush move,i am 35 year old,
Iron deficiency in RLS (e.g. as measured in the potentia nigra) might just be the brain down regulating iron (proven) because of a Thiamine deficiency, which is highly concentrated in a healthy potentia nigra, and is essential for energy and myelin production.
Why are we determining iron status without also looking at the cofactors that are required for iron homeostasis? Could the iron insufficiency simply be a result of not enough copper and retinol in the diet?
@@dryan82 In the meantime I think it is much easier to get into a situation of permanently having too much iron than running out of other nutrients persistently. You can easily correct pretty much any nutrient problem fairly quickly with some multivitamins (with only b12 being slightly problematic), or correct an excess or even heavy metal toxicity by cessation of intake. The only substance this doesn’t work is iron, which effectively permanently stays in the body, with only minimal detox ability. You can improve iron homeostasis, but you can’t change an underlying excess overnight.
Not sure how lower iron levels in the substantia nigra would be the case in RLS now, I’ll have to review. My last guess was that RLS would most likely be a reflex reaction to increase venous blood flow when someone has dvt or the like, since muscles surrounding veins literally pump the veins like a second heart.
There we go: the low iron assumption is probably bogus/misdiagnosis. I got chatgpt, for whatever it’s worth, to agree with me that calcification of the substantia nigra could be mistaken for low iron in TCS/SWI imaging (because both show as hyper intensive) used to conclude in the RLS research paper that there was low iron in the substantia nigra. Really very unlikely as the subs nigra is full of iron and fairly small so shouldn’t hard to ship enough iron there to keep the dopamine production going.
Conversely, iron overload leads to calcification of blood vessels, so this might be the actual problem, especially since normal substantia iron content is already very high.
I can’t find much research on calcified substantia nigra, but that may be why there is a lack of understanding. The pineal gland is well known to very frequently (up to 70% of adults) have calcification issues, and coincidentally is also very high in iron.
“Percentages of calcified pineal shadows from the literature, ranging from 50% to 70%. Study of 265 patients in Fiji, 135 Fijians and 130 Indians, shows a low incidence of calcified pineal shadows, 15·6% in Fijians and 19·2% in Indians.”
Pineal Calcification in Indians and Fijians, 1973
I noticed the other way around. When i ate too much spinach over a week or two, thats when i got RLS. Then months later i ate two bowls of lentils in one day as opposed to my crap diet. I got RLS that night although not too extreme. Does this make sense?
Veggies often contain anti nutrients that compete/inhibit absorption of iron.
Been suffering (lightly) from this since 10 yrs old.
For almost 2 yrs dr required to take blood thinners and blood pressure pills and this triggered my rls to a very extreme condition.
I wonder if cannabis can help?
Hi I have been suffering with RLS for decades. And I tried cannabis and it did not work. Unfortunately the only thing those work are opiates.
Which protein powder are you suggest for RLS patients??
Which Amino acid is good for RLS patients
Doctor . I am working as barber . From seven Months I have problems in my leg . When I work as standing hairs cuting . When I have so much work I feel my legs going to sleep heavy before both legs now left leg only. What I do
Hi Salman! Thank you for your comment. Unfortunately, we are not a doctor's office, but we can provide you with great information and help you find a doctor in your area. Please email info@rls.org.