I know you posted this forever ago, but thank you so much for explaining so clearly. I have a TBI and have related auditory processing issues. I could not understand what my neuroscience professor was saying in any of the online lectures (poor audio, mumbling, rambling) and my accommodation for a written format was denied (there's no textbook either). This really helped me! And I'm relieved to see that you have a bunch of other videos covering some of the other topics in the course
Hi Margaret, Most cases of Bell's Palsy starts to resolve within three weeks, but can last as long as six months. I would advise you, if you haven't already, to see your health provider to properly diagnose and follow your case. Best regards, LB
Hi, Thanks for your kind words and your question! A lesion of the actual nucleus of CN VII (or any motor cranial nerve) will result in a lower motor neuron lesion. A good way to think about it is that the cranial nerve motor nuclei are the cell bodies of the lower motor neurons that control the face and head. (continued)
Amazing video and Doctor!!thanks for simple and narration and great pics!! I have a question:if the lesion is in the VII CN NUCLEUS, is it categorized in upper or lower motor neuron? and which kind of deficit will be resulted? or in general: Which kind of deficit will be resulted if the lesion be between the two YELLOW LINES that you use to show and divide UMN and LMN lesion? thanks for your kindness and help.
These cranial nerve motor nuclei are analagous to the anterior/ventral horn cells that live in the ventral horn of the cord and are the cell bodies of the lower motor neurons which supply skeletal muscles. Damage to the anterior/vental horn cells produce lower motor neuron lesion presentations as well.
So in case of a UML of the corticobulbar tract, will there be spastic paralysis or just paralysis of mid/lower contralateral affected side? I understand spasticity of extremities when corticospinaltract is involved due to loss of inhibition of gamma neurons, but how does that work in the brainstem?
Hi, If it's a corticobulbar lesion, then the paralysis or paresis will be spastic. Flaccid paralysis will occur if the lesion is in the motor nucleus in the brainstem or in the lower motor neuron fibers exiting the nucleus. LB
Hi samed12388, I have never heard of "Racket Sign." "Rocket Sign" is associated with frontal lobe dysfunction in patients with progressive supranuclear palsy. This occurs if the patient rises quickly from a sitting position to a standing position which causes the patient to fall back to the sitting position. This occurs because of postural instability. I am very interested in a reference for Racket Sign so I may include it in the tutorial. Best regards, NeuroDocUniverse
I was diagnosed with bell's palsy however I don't have full paralysis. I can move my cheek and smile a bit, raise my eyebrow even though it is lower than my non damaged side. Is this normal? Do I have bell's palsy or was I misdiagnosed?
I have been diagnosed with Bells palsy but it won't go away...11years this past June. Are they're any studies that need patients? I would like to know if I what's going on with me because I now close my right eye without my left closing...🤔
i had bells palsy for 3 weeks( from 9th Jan. 2013) i am now almost back to normal, my eye does close but not completely my mouth is back to normal, the pain in the ear bothers me though, it is on and off will it ever go away completely??
+mrgetrealpeople Hi there. Thanks for your question. No facial pain is associated with Bell's palsy. Bell's affects CN VII (facial nerve) which is motor to the face and special sensory to the anterior 2/3 of the tongue for taste. Facial pain would be associated with CN V (trigeminal nerve) which supplies sensation to most of the face and anterior areas of the head. An example of a CN V condition associated with pain to the face is trigeminal neuralgia, also known as tic douloureux.
the narrator is an absolute legend. if only all neurology teachers were this good!
Studying for Step...this was WONDERFUL!!!! I never actually 'got this' concept during my neuro block. Now I do!!
Awesome!
You have explained this topic beautifully with perfect diagrams !
So glad it was helpful!
@@NeuroDocUniverse subscriber gained ! You cover a lot of great topics.
O MY GOD I SPENT SEVERAL HOURS TO UNDERSTAND THIS BUT U JUST MAKE IT VERY EASY IN LESS THAN 15 MINUTES.. THANK U SO MUCH......
Thank you very very much...you explain the concept very clear, way better than my instructor.
EXCELLENT! EXCELLENT! EXCELLENT! Clear explanation! Great illusterations that are easy to understand! Wonderful voice! Thank you!!!
Thank you for this wonderful explanation . I think I will never forget this piece of information again.. You are a gem..
+Kais Sh Thanks so much for the kind words.
Studying for my USMLE and found it very helpful. Thank you.
Excellent!!! Good luck with your exam!!!
I know you posted this forever ago, but thank you so much for explaining so clearly. I have a TBI and have related auditory processing issues. I could not understand what my neuroscience professor was saying in any of the online lectures (poor audio, mumbling, rambling) and my accommodation for a written format was denied (there's no textbook either). This really helped me! And I'm relieved to see that you have a bunch of other videos covering some of the other topics in the course
I'm glad you found it helpful!
simple & precise explanation of a very confusing concept... thanku so much !!!
The best explanation for this pathology. Thanks
Very Good And simple explanation just what i have been looking for
This video is amazing...simple with distilled clarity. Thank you
thanx a lot for such a clear narration.
Hi Margaret,
Most cases of Bell's Palsy starts to resolve within three weeks, but can last as long as six months. I would advise you, if you haven't already, to see your health provider to properly diagnose and follow your case.
Best regards,
LB
Thanks for the kind words!
i feel smarter, thank you. Clarified it very well
Beautiful explanation - thanks for taking the time to upload!
Thanks a lot for this awesome explanation. This is very didactic and you have a very pleasant voice. Great upload!
Wow this is absolutely brilliant. Thank you so much. This is such a great video.
You're welcome! Glad it helped you :-)
This was a really clear explanation. Thanks so much!
You're welcome! Glad it helped!
Awesome! You're welcome!
Hi,
Thanks for your kind words and your question!
A lesion of the actual nucleus of CN VII (or any motor cranial nerve) will result in a lower motor neuron lesion. A good way to think about it is that the cranial nerve motor nuclei are the cell bodies of the lower motor neurons that control the face and head. (continued)
Thanks a lot, great piece of work
Amazing video and Doctor!!thanks for simple and narration and great pics!!
I have a question:if the lesion is in the VII CN NUCLEUS, is it categorized in upper or lower motor neuron?
and which kind of deficit will be resulted?
or in general: Which kind of deficit will be resulted if the lesion be between the two YELLOW LINES that you use to show and divide UMN and LMN lesion?
thanks for your kindness and help.
An excellent educational video! Thank you so much.
Very clear and concise! Thanks for the explanation!
I'm glad this video was helpful. You are so welcome!
Wonderful..deserves more views n appreciation
Wow, GREAT VIDEO!! This is the best explanation yet!! I truly appreciate it!! THANK YOU!!
Thank you for a lovely clear explanation
Great.... thanx a lot for such a nice explanation 👌
These cranial nerve motor nuclei are analagous to the anterior/ventral horn cells that live in the ventral horn of the cord and are the cell bodies of the lower motor neurons which supply skeletal muscles. Damage to the anterior/vental horn cells produce lower motor neuron lesion presentations as well.
very good learning process
Great job. Thank you for education
Incredible explanation thank you!
this was of great help..thank you very much
Very well explained! And I love your voice, it is amazing!
So in case of a UML of the corticobulbar tract, will there be spastic paralysis or just paralysis of mid/lower contralateral affected side?
I understand spasticity of extremities when corticospinaltract is involved due to loss of inhibition of gamma neurons, but how does that work in the brainstem?
Wow, that was incredible!
Wow ...very well explained , you have a soothening voice too..:) at last I understood it
wow, this is so satisfactorily explanatory.. thanks so much!!!! :)
Hi,
If it's a corticobulbar lesion, then the paralysis or paresis will be spastic. Flaccid paralysis will occur if the lesion is in the motor nucleus in the brainstem or in the lower motor neuron fibers exiting the nucleus.
LB
Thank you!
You're welcome!
wow what a explanation , thank u so much .
Upper motor neuron damage leads to lower face paralysis also called racket sign(orbicularis oris) and will not affect the nose or eye
good explanation
Hi samed12388,
I have never heard of "Racket Sign." "Rocket Sign" is associated with frontal lobe dysfunction in patients with progressive supranuclear palsy. This occurs if the patient rises quickly from a sitting position to a standing position which causes the patient to fall back to the sitting position. This occurs because of postural instability.
I am very interested in a reference for Racket Sign so I may include it in the tutorial.
Best regards,
NeuroDocUniverse
plz mention the sides which gets affected in bell's palsy n facial palsy?ipsilateral or contralaterally.
Ipsilateral
Thank you so much!
so awesome...thanks
+Mehrzad Moheb You're welcome!
with the top part of the nucleus you mean the dorsal portion of the facial motor nucleus?
Would a corticobulbar tract lesion result in spastic or flaccid? I was thinking spastic since they're UMNs?
Thanks
I was diagnosed with bell's palsy however I don't have full paralysis. I can move my cheek and smile a bit, raise my eyebrow even though it is lower than my non damaged side. Is this normal? Do I have bell's palsy or was I misdiagnosed?
thank u very much
thanks it really helped
I have been diagnosed with Bells palsy but it won't go away...11years this past June. Are they're any studies that need patients? I would like to know if I what's going on with me because I now close my right eye without my left closing...🤔
Tnx for sharing!!
very useful.. thx😍
i had bells palsy for 3 weeks( from 9th Jan. 2013) i am now almost back to normal, my eye does close but not completely my mouth is back to normal, the pain in the ear bothers me though, it is on and off will it ever go away completely??
thank you for simplifying that :)
So if my eye closes (90% of the lid-but no eyebrow movement) but the rest of that side is paralyzed , where in the nerve is the issue?
It's difficult to tell without a thorough exam, but if there is only partial lid closing the problem can still be the facial nerve (CNVII).
thank u
You're welcome!
You're welcome!!!
Thanks!!! Glad it was helpful..
finally get it!
Sure! Camtasia software and a Wacom Intuos tablet.
Do you have face pain from this condition on the side affected?
+mrgetrealpeople Hi there. Thanks for your question. No facial pain is associated with Bell's palsy. Bell's affects CN VII (facial nerve) which is motor to the face and special sensory to the anterior 2/3 of the tongue for taste. Facial pain would be associated with CN V (trigeminal nerve) which supplies sensation to most of the face and anterior areas of the head. An example of a CN V condition associated with pain to the face is trigeminal neuralgia, also known as tic douloureux.
I have a vascular loop making contact TGN
And it drups my face..........
thank you ..great explanation =)
thanks love
You're so welcome :-) !
I LOVE YOU!!!!
THANK U :)
U r amazing!!😘😘
really nice
the patient with Bell "s palsy won't be able to blink and to close his eye completetly on the affected side
plz guys .....my fathere have MND ....i ask u to help me to get cure for it plz... my father dying...
EUREKA!!!!
meenvaruve
l
Thank you!
You're welcome!
You're welcome!
You're welcome!
You're welcome!