Anemia of chronic disease | Hematologic System Diseases | NCLEX-RN | Khan Academy
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- Опубліковано 20 жов 2014
- Created by Nauroz Syed.
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she is wonderful instructor! very well structure course. thanks
Thank you greatly for this valuable information! I am grateful to have found your channel!
This is so perfect and short. Thank you, keep up the good work.
This is very helpful! Thank you!
Fantastic explanation esp the reasoning why the body decreases iron absorption and the role of hepcidin, thank you so much :-)
!! THANKS ..GREAT VIDEO
Wonderful explanation. Thanks a lot
A logical and succinct clear brilliant explanation - Much appreciated.
Great video, easy to understand. Thank you
Awesome explanation, very helpful!
Perfect, thank you so much
Thank you!
So simple n straightforward thank you!!
Thankk youu so muchhhh for this explanation!!!!!
Keep up the good work. Thanks . Great job!
Very clear thank you
Great !!
Thank you 😘
شكرا😍🙈
Thankyou
Thanks doctor
Clear!
Fabulous!
Wonderful explanation
amazing
Beautiful
Well done mam...
Hepcidin is an acute phase reactant
Hepcidin also inhibits EPO
new rbcs made with less heme because there is less available Fe -> incr free erythrocyte protoporphyrin FEP
IDA and ACD is first normocytic then microcytic
thank you. brilliant explanation as always khan academy 🙌🏻
ተባረኪ 😭👍
wonderfullll..... this step is so important as how inflammation causes anemia
Really explains it, I’ve just had an iron infusion because mines really low, having investigations to find out why atm
This has been well explained. And in only 6 minutes my understanding has been greatly enlightened. Thank you!
Wow ❤❤❤❤❤❤
Thx for this great explain it's really helpful ❤❤
very very good
Why then ferritin increase ? And TIBC decrease ???
Nice
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nice video... just one thing you said the macrophages in liver degrade the RBC whereas that happens in the spleen not in the liver please clarify.
Both organs, no? Don't know proportions for each. While in circulation, RBCs slowly accumulate damage that is repaired by macrophages of the spleen. Finally, after 120 days of circulation, senescent RBCs are removed from the circulation by splenic and liver macrophages.
If the hepcidin decrease RBC degradation then what happen to the expired RBC?
This is an excellent explaination. I have anemia of CD. I have cancer also. I also just finished radiation (inflammation?) How do they know which one it is?
How are you holding up
why is ferritin high in anaemia of chronic disease and low in iron deficieny? and why does total iron binding capacity is high in IDA and low in anemia of chronic disease? thank you
ferritin is low in ida since the storage pool of iron gets exhausted from lack of iron, thereby decreasing ferritin (which stores iron), On the other hand, ferritin is high in ACD because the reticuloendothelial system gets activated to increase iron retention. this is quite important in preventing bacteria (which may be present due to inflammation or other conditions) from utilizing the bodys iron.
Does thalessemia leads to anaemia of chronic disease ??
You didn't mention erythropoietin.
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So say someone with cancer had some sort of anemia, are you saying the cancer itself is causing it?
What about the 6-7-8 different lab test the doctor orders weekly is that not causing the anemia?
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great video, but its pronounced "com-pen-sa-tory" NOT "compen-satory"
Nonsense. That is why you get different accents. You do not have to pronounce just one way. Expand your world.
I don't even think most Americans pronounce it like she did.
Also if you agree with the way duodenum was pronounced I don't think we are going to get along :P haha
sit down, be humble. the world is much wider than America.
Doesn't matter how she said it, you understood what she was saying.