Dr. Kulkarni, you just said that there are NOT a lot of good ways to monitor heart disease in asymtomatic individuals. Wouldn't you agree that cardiovascular inflammatory biomarkers (hs-CRP, MPO, Lp-PLA2, Alb/Creat) and AI assisted CCTA's are very helpful in MONITORING a patient's plaque burden, plaque composition (hard vs soft plaque) and degree of coronary endothelial dysfunction/inflammation?
Great question, RohitKumar. Cardiologists and lipidologists keep saying that Lp(a) is very static and changes very little during one's life. However, I am finding more and more evidence that MANY, MANY things can cause extreme variations in a person's Lp(a) levels-trauma, surgery, inflammation, dietary changes, fasting. You may want to take a look at the UA-cam videos on Lp(a) by researchers Dr. Nadir Ali and Ms. Siobhan Huggins.
@@JMK-vo8pv i watched their videos the also told that its genetic beacuse dietary can change level but not too much i dont know what to do has there is a difference between mgdl or nmoll beacuse significant increase cannot be increased by diet
Just like Dr. Nadir Ali says- Lp(a) does NOT CAUSE heart disease/coronary plaque. Lp(a) only shows up in coronary plaque after there has been an injury to the coronary endothelium. Lp(a) is a "firefighter," not an aronist. Therefore, I would not worry about what my Lp(a) level is. Who cares? Lp(a) particles do NOT CAUSE atherosclerosis. @@RohitKumar-ei2dw
Dr. Kulkarni, you just said that there are NOT a lot of good ways to monitor heart disease in asymtomatic individuals. Wouldn't you agree that cardiovascular inflammatory biomarkers (hs-CRP, MPO, Lp-PLA2, Alb/Creat) and AI assisted CCTA's are very helpful in MONITORING a patient's plaque burden, plaque composition (hard vs soft plaque) and degree of coronary endothelial dysfunction/inflammation?
Mine was 225.5 nmol/l when i tested in russia and second time i tested it was 121.7 mg/dl in india if i calcalute mg/dl into nmol/l it has increased
Hello if lipoprotien a is a genetic and levels do not change throughput your lifetime then why it is increasing significantly.
Great question, RohitKumar. Cardiologists and lipidologists keep saying that Lp(a) is very static and changes very little during one's life. However, I am finding more and more evidence that MANY, MANY things can cause extreme variations in a person's Lp(a) levels-trauma, surgery, inflammation, dietary changes, fasting. You may want to take a look at the UA-cam videos on Lp(a) by researchers Dr. Nadir Ali and Ms. Siobhan Huggins.
@@JMK-vo8pv i watched their videos the also told that its genetic beacuse dietary can change level but not too much i dont know what to do has there is a difference between mgdl or nmoll beacuse significant increase cannot be increased by diet
Just like Dr. Nadir Ali says- Lp(a) does NOT CAUSE heart disease/coronary plaque. Lp(a) only shows up in coronary plaque after there has been an injury to the coronary endothelium. Lp(a) is a "firefighter," not an aronist. Therefore, I would not worry about what my Lp(a) level is. Who cares? Lp(a) particles do NOT CAUSE atherosclerosis. @@RohitKumar-ei2dw