Thank you Dr. Hanna! You are a gifted teacher and this is the best and most practical narrative on bifurcation stenting. Will be anxiously waiting for the sequel on this topic.
There is another provisional stent strategy not discussed. We students named this strategy after our beloved mentor who would usually say after he snow ploughed a relatively small branch… “ You didn’t need that branch “
It is a phenomenal talk as usual. We are lucky to have Dr. Hanna at University of Iowa. We look forward to the new edition of your amazing book. Thank you for being a great physician and teacher
May Allah swt bless you for being so generous and making it easier for others to learn. Waiting for talk cullotte and DK crush techniques and PCI of LMS. I pray that your teaching sessions will raise your darajaat snd will be sadqa i jaariyah for u.
Best! please keep on making more conceptual cardiology lectures. Studying and enjoying from ELIAS B HANA 2nd edition book probably the BEST CARDIOLOGY BOOK
Sir stay blessed. Sir kindly make other videos for intervention and general cardiology also basics tips and tricks. It’s really make my day. Sir any book of your regarding intervention cardiology. ???
thank you so much for your great lecture. Can you elaborate the reason why PLAQUE SHIFT produces more severe outcome compared to CARINA SHIFT, since previously I thought that plaque shift during main branch stenting will be easy to break down while carina shift creates a new form of solid stent therefore not functionally
A nice study published in CCI showed that CARINA shift is rarely associated with FFR compromise, while PLAQUE shift, which is less common than carina shift, is the one that causes functional FFR compromise. onlinelibrary.wiley.com/doi/epdf/10.1002/ccd.24956 The likely reasons are 3-fold: (1) carina shift is a very focal process (functional effect correlates with how long the lesion is, beside how tight); (2) Carina shift is an eccentric geometric distortion: the lumen is not as narrow in 3 D as it may appear in one view; (3) possibly, vasoreactivity is more preserved when there is less plaque. Imagine carina shift as squashing of the SB ostium and changing it from a circle to an ellipse: the overall area is less reduced than if you shift plaque. This is good news, as carina shift is more easily prevented by: (i) avoid MB stent oversizing (side to distal vessel); (ii) POT of proximal MB stent It is all about plaque burden. Plaque burden of MB and SB predicts plaque shift. This explains why in the definition of complex true bifurcation (higher risk of SB occlusion), plaque burden is key: length of SB disease>10 mm, heavy plaque burden in the main vessel, heavy calcium.
Thank you Dr. Hanna! You are a gifted teacher and this is the best and most practical narrative on bifurcation stenting. Will be anxiously waiting for the sequel on this topic.
🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳👍🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳🥳
There is another provisional stent strategy not discussed. We students named this strategy after our beloved mentor who would usually say after he snow ploughed a relatively small branch… “ You didn’t need that branch “
😂😂😂😂😂😂😂😂
MASHALLAH, what an amazing lectures, never seen before. Really Great sir🙏
It is a phenomenal talk as usual. We are lucky to have Dr. Hanna at University of Iowa. We look forward to the new edition of your amazing book.
Thank you for being a great physician and teacher
brilliant presentation. very lucid and practical. Always on the look out for your talks
Thank you Professor Hanna, amazing talk!
As usual; another excellent presentation, passionately waiting for your talk regarding CTO intervention
A marvelous lecture from an outstanding professor. Thanks alot
Amazing video as always Dr. Hanna! Looking forward to the new edition of your textbook. Already read the last one and it was great.
Mashallah you are a great teacher
one of the best teachers
May Allah swt bless you for being so generous and making it easier for others to learn. Waiting for talk cullotte and DK crush techniques and PCI of LMS.
I pray that your teaching sessions will raise your darajaat snd will be sadqa i jaariyah for u.
Amazing Video. Thank you Dr. Hanna
Thanks a lot. Very informative. Keep posting
Thanks a lot
Great teaching !
Please continue
As always excellent sir….👌👌👌
Great presentation again👏🏻👏🏻👏🏻👏🏻
Thanks a lot very illustrative presentation
Thank you for sharing this information
in provisional techniqe what do you think for avoid wire rapture do rewiring before pot
Excellent video..thank you very much
Best! please keep on making more conceptual cardiology lectures. Studying and enjoying from ELIAS B HANA 2nd edition book probably the BEST CARDIOLOGY BOOK
Best lectures
Sir can you plz share the presentarion
I loved it … very useful
Sir stay blessed. Sir kindly make other videos for intervention and general cardiology also basics tips and tricks. It’s really make my day. Sir any book of your regarding intervention cardiology. ???
Big Fan of you sir😍
Excellent
I wished to have u as a Mentor in my Hospital in Germany
We all do!
how can i connect with you? @nikesnipe
@@rezaulislam8953 hello!
Dr.Hanna great lecture! is it possible to download text presentations? it would help a lot because English is not my primary language.
thank you sir a million
Thank you sir
great. thanks !!!
thank you
thank you so much for your great lecture. Can you elaborate the reason why PLAQUE SHIFT produces more severe outcome compared to CARINA SHIFT, since previously I thought that plaque shift during main branch stenting will be easy to break down while carina shift creates a new form of solid stent therefore not functionally
A nice study published in CCI showed that CARINA shift is rarely associated with FFR compromise, while PLAQUE shift, which is less common than carina shift, is the one that causes functional FFR compromise. onlinelibrary.wiley.com/doi/epdf/10.1002/ccd.24956 The likely reasons are 3-fold: (1) carina shift is a very focal process (functional effect correlates with how long the lesion is, beside how tight); (2) Carina shift is an eccentric geometric distortion: the lumen is not as narrow in 3 D as it may appear in one view; (3) possibly, vasoreactivity is more preserved when there is less plaque. Imagine carina shift as squashing of the SB ostium and changing it from a circle to an ellipse: the overall area is less reduced than if you shift plaque. This is good news, as carina shift is more easily prevented by: (i) avoid MB stent oversizing (side to distal vessel); (ii) POT of proximal MB stent
It is all about plaque burden. Plaque burden of MB and SB predicts plaque shift. This explains why in the definition of complex true bifurcation (higher risk of SB occlusion), plaque burden is key: length of SB disease>10 mm, heavy plaque burden in the main vessel, heavy calcium.
@@eliashanna8248 it's so great to hear this explanation. I learned a lot and really appreciate your explanation
Dr thx for great presentation again... can take
it pdf or send to me
Great 8
Done
I beg ALLAH to bless your age, life, and health.