I was told if you have optic nerve damage its too late. you will always end up with blindness. especially if the mri shows there is nothing indicating why the nerve is being damaged. even no proof of tuberculous. even with perfect blood work. so sad
These below outcomes now attributed to the procedure electroshock in my medical records. I was told ECT was safe. I live with these damages as so many others do to one degree or another. This is TBI. This is full body trauma from an electrical mechanism, with damages that can evolve years out to include CTE. I was recently seen by a provider (neuro/opthomologist) who said I had “convergence insufficiency” and would not even address or put in his own notes this mechanism for fear of reprisals by peers most likely. I was discounted, minimized, bullied, and treated very poorly by this neuro/ophthomologist who in no way wanted to let the cat out of the bag, no matter how it impacted me as the patient. When I asked him to address the damages and causation that would assist me with rehabilitative measures he minimized and discounted these obvious outcomes so he would not have to make a stand in his notes to this very apparent harm taking place. We deserve help and kindness in our compromised state. Not to be subjugated to further abuse after what we have already been through with “trusted” providers. We are PTSD survivors as a result and this is not any way to treat a patient. At end of this appointment that got very heated, I was left with no help from him and asked to leave. This is no way to treat an individual. I was never disrespectful, but also knew the issues involved and did not let him "off the hook." Electroshock patients are going to start to see various providers for assistance and I ask that we be met with compassion despite the reputations and monies involved. Problem is everyone seems to have monetary or employment/position gains to be made if they keep the lid on this no matter how many are hurt. Please remember you are healers for the good of your patient. We are greatly suffering while you all pretend otherwise. California courts have proved brain injuries you are already well aware of based on your training. No advanced tests were done outside of a very basic optho exam my primary did, and no imaging or rehabilitative services. Don't test, don't have to worry about addressing damages and causes in notes. Phonophobia. Visual and auditory stimulation problems. Seriel 7’s markedly impaired Mild L ptosis R hypertropia worse in L lateral gaze L exophoria L upper and R lower facial paresis L roll had tilt Olfactory recognition impaired bilateral VA ration horizontal square wave jerks R:2:1 L: down-beat nystagmus 2:1 Saccade testing reveals latencies increased all planes except U/L Marked cervical substitutions with pursuits in all planes with intrusive saccades worsening in L prusuites Pursuits downward reveal intorsional glissades Gait testing reveals mild decrease inR arm swing: with dual tasking, gait becomes slightly wide-based and arm swing slightly decreases. Finger-nose past pointing R>L Somatic pinwheel perception diminished L L5 Vestibular head impulse testing: Moderately decreased in LARP plane Saccadometry: Prosaccade 20 degree : intrusive saccades to the R Anti saccade 10 degree: 79 percent directional error rate Nystagmus: High frequency right beat and down beat nystagmus Central gaze: Head movement, L pstosis and nystagmnus Horizontal gaze L 24 degree Notable pitch plane head movement Horizontal gaze R 24 degree: Increased fatigue, decreased stability Upward gaze 14 degree: Notable pitch plane head movement Downward gaze 14 degree: Notable pitch polane head movement. Horizontal optokinetics 25 dps: L optokinetics provoked dysconjugate gaze. Reflex failed with R otokinetics Horizontal optokinetics with volitional targeting: Worsens Vertical pursuits 10 degrees: Intrusive saccades with downward pursuits Random vertical saccades: Upward intrusive saccades, cannot maintain downward gaze Vertical optokinetics 25 dps: Reflex failed. Vertical optokinetics with volitional targeting: Worsens Repeated random horizontal saccades; Latencies increased significantly bilaterally Stop this battery of patients. You have a duty to warn, protect, and not cause harm. Are you participating in this either actively or passively? Someone please stand up for patients! Please see ectjustice now owned by law firms participating in national product liability suit around devices.
Can Bilateral Amblyopia cause this condition to occur? I have the Bilateral Amblyopia and both my grandmother on y father's side, his Mom and his brother both went blind in their 50s and I am in my 50s and already have some vision loss at night time when there is no light I go blind and when light is very minimul, I see very minimul.
Please doctor ineed help for my
I was told if you have optic nerve damage its too late. you will always end up with blindness. especially if the mri shows there is nothing indicating why the nerve is being damaged. even no proof of tuberculous. even with perfect blood work. so sad
These below outcomes now attributed to the procedure electroshock in my medical records. I was told ECT was safe. I live with these damages as so many others do to one degree or another. This is TBI. This is full body trauma from an electrical mechanism, with damages that can evolve years out to include CTE.
I was recently seen by a provider (neuro/opthomologist) who said I had “convergence insufficiency” and would not even address or put in his own notes this mechanism for fear of reprisals by peers most likely. I was discounted, minimized, bullied, and treated very poorly by this neuro/ophthomologist who in no way wanted to let the cat out of the bag, no matter how it impacted me as the patient. When I asked him to address the damages and causation that would assist me with rehabilitative measures he minimized and discounted these obvious outcomes so he would not have to make a stand in his notes to this very apparent harm taking place. We deserve help and kindness in our compromised state. Not to be subjugated to further abuse after what we have already been through with “trusted” providers. We are PTSD survivors as a result and this is not any way to treat a patient.
At end of this appointment that got very heated, I was left with no help from him and asked to leave. This is no way to treat an individual. I was never disrespectful, but also knew the issues involved and did not let him "off the hook."
Electroshock patients are going to start to see various providers for assistance and I ask that we be met with compassion despite the reputations and monies involved. Problem is everyone seems to have monetary or employment/position gains to be made if they keep the lid on this no matter how many are hurt. Please remember you are healers for the good of your patient. We are greatly suffering while you all pretend otherwise. California courts have proved brain injuries you are already well aware of based on your training.
No advanced tests were done outside of a very basic optho exam my primary did, and no imaging or rehabilitative services. Don't test, don't have to worry about addressing damages and causes in notes.
Phonophobia. Visual and auditory stimulation problems.
Seriel 7’s markedly impaired
Mild L ptosis
R hypertropia worse in L lateral gaze
L exophoria
L upper and R lower facial paresis
L roll had tilt
Olfactory recognition impaired bilateral
VA ration horizontal square wave jerks R:2:1 L: down-beat nystagmus 2:1
Saccade testing reveals latencies increased all planes except U/L
Marked cervical substitutions with pursuits in all planes with intrusive saccades worsening in L prusuites
Pursuits downward reveal intorsional glissades
Gait testing reveals mild decrease inR arm swing: with dual tasking, gait becomes slightly wide-based and arm swing slightly decreases.
Finger-nose past pointing R>L
Somatic pinwheel perception diminished L L5
Vestibular head impulse testing: Moderately decreased in LARP plane
Saccadometry: Prosaccade 20 degree : intrusive saccades to the R
Anti saccade 10 degree: 79 percent directional error rate
Nystagmus: High frequency right beat and down beat nystagmus
Central gaze: Head movement, L pstosis and nystagmnus
Horizontal gaze L 24 degree Notable pitch plane head movement
Horizontal gaze R 24 degree: Increased fatigue, decreased stability
Upward gaze 14 degree: Notable pitch plane head movement
Downward gaze 14 degree: Notable pitch polane head movement.
Horizontal optokinetics 25 dps: L optokinetics provoked dysconjugate gaze. Reflex failed with R otokinetics
Horizontal optokinetics with volitional targeting: Worsens
Vertical pursuits 10 degrees: Intrusive saccades with downward pursuits
Random vertical saccades: Upward intrusive saccades, cannot maintain downward gaze
Vertical optokinetics 25 dps: Reflex failed.
Vertical optokinetics with volitional targeting: Worsens
Repeated random horizontal saccades; Latencies increased significantly bilaterally
Stop this battery of patients. You have a duty to warn, protect, and not cause harm. Are you participating in this either actively or passively? Someone please stand up for patients!
Please see ectjustice now owned by law firms participating in national product liability suit around devices.
Can Bilateral Amblyopia cause this condition to occur? I have the Bilateral Amblyopia and both my grandmother on y father's side, his Mom and his brother both went blind in their 50s and I am in my 50s and already have some vision loss at night time when there is no light I go blind and when light is very minimul, I see very minimul.
I know a doctor who can help you
Contact please