The Neurobiology & Phenotypes of Trauma And It's Implications for Clinical Practice

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  • Опубліковано 7 вер 2024
  • In this video, we unravel the intricate world of PTSD phenotypes, exploring the neurobiology of trauma and its implications for clinical practice. Discover the key brain centers involved, from the threat-focused amygdala to the emotional hub of the anterior cingulate cortex. Learn how individualized treatment plans can be crafted based on the recognition of different phenotypes. Ready to delve into the complexities of PTSD?
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КОМЕНТАРІ • 13

  • @jacklinemmochi6433
    @jacklinemmochi6433 7 місяців тому +1

    Great

  • @yamyite
    @yamyite 6 місяців тому +1

    I’ve watched a few of your videos and they cover PTSD briefly in this video and Akathisia vs RLS.
    Could you please do a video on long term high dose opiate use and PTSD, night terrors, RLS. Brain recovery? I’ve heard parts of the brain turn black and die off, is this true? Therefore homeostasis within the brain is therefore unobtainable? Is there a tipping point that the brain can’t recover from, especially if parts do indeed turn necrotic?
    Many thanks

    • @PsychiatrySimplified
      @PsychiatrySimplified  6 місяців тому +1

      That’s very specific. The turn black and die off is not true - in stroke for example there can be cell death but depending on the extent recovery can occur . Neuroplasticity is present - you can see my video on neuroplasticity. Even in clinical practice we see situations where the brain may have significant atrophy but with treatment they can reach a significant level of function or normal function. In relation to your question of tipping point - it’s very individual - in some a small lesion can be devastating - depends on the area involved and the neuroplasticity changes that occur after while in others even a large lesion can result in significant recovery.

    • @yamyite
      @yamyite 6 місяців тому +1

      @@PsychiatrySimplified ok thank you 🙏

  • @Elizabethpepper8
    @Elizabethpepper8 8 місяців тому +1

    Is it possible to rotate between two? When triggered extreme reactions, verse when not detachment.

  • @Truerealism747
    @Truerealism747 8 місяців тому +2

    Have you got any insight of now ime diagnosed heds aspergers add fybromyalgia called trifecta

    • @PsychiatrySimplified
      @PsychiatrySimplified  8 місяців тому

      Yes there is also a Pentad - add POTS and MCAS to this. See the video on POTS.

  • @mobillegenuiya4586
    @mobillegenuiya4586 8 місяців тому

    So my frontal lobe is slowing shown in EEG, recently my brain getting back to the cognitive function it used to be (great indication that it is recovering) but i lose 3 kg in 2 weeks, is it normal that brain recovery make you lose weight?

  • @mobillegenuiya4586
    @mobillegenuiya4586 8 місяців тому +1

    Can frontal lobe dysfunction cause tongue fasciculation?

    • @PsychiatrySimplified
      @PsychiatrySimplified  8 місяців тому

      Yes movement disorders can be associated with frontal lobe dysfunction

  • @joaquinvalentini6639
    @joaquinvalentini6639 8 місяців тому +1

    Hello, greetings from Argentina.
    Is there any difference in the treatment approach between both phenotypes?

    • @PsychiatrySimplified
      @PsychiatrySimplified  8 місяців тому +1

      Great question - emotional undermodulation - focus on reduction of hyperarousal and then strengthen PFC . The emotional overmodulation type - is also the same as first one except that one has to recognise that reduction of hyperarousal can worsen dissociation in some - hence why other strategies ( motor tone , fluid increase ) are considered. Agents such as cannabinoids and naltrexone are considered in the latter emotional overmodulation phenotype. Read more here 👉psychscenehub.com/psychinsights/the-neuroscience-of-dissociation/