if the normal reflex is to cyclotorsion away from the head tilt (to the right in his initial example 0:21), and a lesion stimulates/simulates that same head tilt, why would the eyes now cyclort to the left? 3:35
I watch your videos just as a pastime and learn something lovely each time. I sat for mrcp part 2 and I had watched this video before and got a question correct! Wish you a long and happy life ❤
Hi Dr. Lee, wonderful video! Could you explain in more detail why there is bilateral torsion to the hypotrophic eye? and how the brain interprets a lesion in the VOR pathway? Thank you!
Hello doctor. How come defects in the pathway cause stimulation. Which means this pathway interacts with vertical gaze centres through concomitant excitation and inhibition. So interruption leads to disinhibition and so excitation?
Normally, the vestibular system has a constant electrical potential at rest. When there is damage to the vestibular system on one side, the potential on lesion side decreases, resulting in "imbalance". What happens then, as described in the doc
Can you do a video on what could cause a comorbid third nerve palsy, with ptosis, Apraxia, diplopia, and alternating intermittent exotropia and anisocoria? More specifically - rarer causes when "neurodegenerative" diseases are ruled out and MRI's/CTs are "normal." Not asking for a diagnosis. And don't worry, definitely not asking for a family member who's stumped multiple neuro-opthalmologist. 😵💫🫠 Just a curious George over here🥺
Andrew gave a super interesting levture last year at the the meeting of the Swedish Ophtamology This is a super interesting and on point piece!
Fantastic explanation. And perfectly explained the 4th and 5th steps for ruling out IV palsy vs skew. 👏
if the normal reflex is to cyclotorsion away from the head tilt (to the right in his initial example 0:21), and a lesion stimulates/simulates that same head tilt, why would the eyes now cyclort to the left? 3:35
I watch your videos just as a pastime and learn something lovely each time. I sat for mrcp part 2 and I had watched this video before and got a question correct! Wish you a long and happy life ❤
❤❤❤❤ may god BLESS YOU ALWAYS! Best!
The contrast is so high my pupils have never been smaller, Doctor Brotherman.
You had me at interstitial nucleus of cajal!
Hi Dr. Lee, wonderful video! Could you explain in more detail why there is bilateral torsion to the hypotrophic eye? and how the brain interprets a lesion in the VOR pathway? Thank you!
Hello doctor. How come defects in the pathway cause stimulation. Which means this pathway interacts with vertical gaze centres through concomitant excitation and inhibition. So interruption leads to disinhibition and so excitation?
Normally, the vestibular system has a constant electrical potential at rest. When there is damage to the vestibular system on one side, the potential on lesion side decreases, resulting in "imbalance". What happens then, as described in the doc
Nice video! What does the Macaque Monkey indicate in the video 2:02
I believe it’s a funky menomic 😂 as this is the jocker?
Can you do a video on what could cause a comorbid third nerve palsy, with ptosis, Apraxia, diplopia, and alternating intermittent exotropia and anisocoria? More specifically - rarer causes when "neurodegenerative" diseases are ruled out and MRI's/CTs are "normal." Not asking for a diagnosis. And don't worry, definitely not asking for a family member who's stumped multiple neuro-opthalmologist. 😵💫🫠 Just a curious George over here🥺
fantastic
perfectly explained
Applauso