AWESOME, that is what I expect from a professor. My book has all the information but you have organized it so well that one can compare and contrast, making it easier to understand, more importantly remember. Thank you!!!
Excellent job..well organized and a great overview. FYI though, HEMOstasis is clot formation & HOMEOstasis is the regulation of the entire body to normal.
1:00:00 = there is mistake that said vitamin k can synthesize 2,7,11,10.....it should be 9 instead of 11................................. Vitamin K can make factors 2,7,9,10 protein C and S............................ and it cannot synthesize factors but it can make it proper functioning factors by attaching its carboxylic group to these factors.
I'm misunderstanding one point @6:00, what does red blood cell damage have to do with primary hemostasis (subendothelial collagen exposure, platelet attachment, activation and degranulation)? This sounds more pathognomic of "endothelial" cell damage instead of RBCs.
my sister was taken by this nasty desease last place for these cells to multiply was inside her spine heading for the brain unfortunately she passed away 3 years ago no surgeon would perform any operation sadly there went a life God bless her
Mr. Woodruff. I think I used to be your student in Hanford. Aren't you teaching in Visalia now? Didn't you start teaching at either Fresno state or College of Sequoias?
I don't know if you're the same person but you look like him. That was way back in the late 70's and early 80's when I think I knew you at the school. I don't know. You can let me know.
I’m so glad I found your lectures. They’re the live, rev-me-up complement to my online pathophys class and you really help me get stuff. Thank you!
AWESOME, that is what I expect from a professor. My book has all the information but you have organized it so well that one can compare and contrast, making it easier to understand, more importantly remember. Thank you!!!
you help me understand topics in a simplier way. thank you. you are helping me in my masters.
Excellent job..well organized and a great overview. FYI though, HEMOstasis is clot formation & HOMEOstasis is the regulation of the entire body to normal.
simplified lecture , worth watching, thank you sir,, may God bless you
I love your lectures. You make them interesting, encourage thinking and have a nice voice.
Wow, thank you!
Great video! Extremely well put together and clear.
The best lecture ever! thank you very much :-)
Thank you for this. I'm taking pathophysiology online and it gets confusing. You made it clear and in order.
Very articulated lecture :) thanks. I learned more in 1hour then i did in two months
Same here
What an awesome lecture! Thank you!!
Doctor, thanks for the informative video. What is the soundtrack you used for you question ?
this lecture is awesome!!
Great Lecture!
wow, long and very informative.. excellent.. thanks
Great lecture !! Thank you !!
Thanks, I learned a lot!
I've studied all this with full complex details. I recommend Harmenning book for hematology.
I am taking this class now and I learn more with your lectures than reading my textbook. Thank you so much!
Very Good lecture! Thank you.
Thank you it’s very helpful 🙏
Excellent job
exceptional presentation!! but isn't anaemia defined exclusively by hemoglobin ?
Very good enjoying listening
awesome, knowledgeable!!!
Thanks to make a video sir
1:00:00 = there is mistake that said vitamin k can synthesize 2,7,11,10.....it should be 9 instead of 11.................................
Vitamin K can make factors 2,7,9,10 protein C and S............................
and it cannot synthesize factors but it can make it proper functioning factors by attaching its carboxylic group to these factors.
At 8:49, it should be Plasminogen --> Plasmin --> FDP
Thank you for pointing this out! Plasminogen (inactivated)-->Plasmin (activated)-->cleaves fibrin to dissolve it (FDP or fibrin dissolving product)
I'm misunderstanding one point @6:00, what does red blood cell damage have to do with primary hemostasis (subendothelial collagen exposure, platelet attachment, activation and degranulation)? This sounds more pathognomic of "endothelial" cell damage instead of RBCs.
Michael Savilla I think it was supposed to be "endothelial cell"
Edit: Didn't see the whole comment at first.
Cheers
Agree!
Great lecture
Nice lecture
amazing
my sister was taken by this nasty desease last place for these cells to multiply was inside her spine heading for the brain unfortunately she passed away 3 years ago no surgeon would perform any operation sadly there went a life God bless her
Mr. Woodruff. I think I used to be your student in Hanford. Aren't you teaching in Visalia now? Didn't you start teaching at either Fresno state or College of Sequoias?
Where are all of these review questions coming from? A previous lecture?
I don't know if you're the same person but you look like him. That was way back in the late 70's and early 80's when I think I knew you at the school. I don't know. You can let me know.
thank you, thank you, thank you!!!
instructive
42:00 Great so a patient with sickle cells anemia can have a chest pain presented as chest pain (was having a hard time getting that information)
Thank u very very much 🙏
Most welcome 😊
Is this based on Robbins?
Nice
nice
tnx and excellent
Hemostasis is different than homeostasis.
ok
great lecture
𝗔𝗺𝗮𝘇𝗶𝗻𝗴!!
is not good 🤦🏻♂️💔