HY USMLE Q

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This is Audio Qbank Q #921
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[@Mehlmanmedical]

Our New USMLE Telegram group (link valid at least at time of this clip): t.me/+mSDYK3fV2wdkNmY0
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Mehlman HY Cardio PDF: mehlmanmedical.com/hy-cardio/
Mehlman HY Renal PDF: mehlmanmedical.com/hy-renal/

[@yueh2024]

Love this!

[@alanwaterman1328]

Should we always think serum Na is a down arrow in congestive heart failure even though we can over think with a few competing mechanisms in play. For instance increase in ANP will inhibit ENaC channel and lead to increased sodium excretion hence, water follows Na and diuretic effect and increase serum Na. Compared to baroreceptor increasing ADH and therefore hyponatremia and decreased renal perfusion and activate RAAS and hyponatremia. Does RAAS and baroreceptor always win?

[@Mehlmanmedical]

Family Med form for 2CK asks why there's hyponatremia in patient with heart failure. Answer on the form is "baroreceptor-mediated ADH release." Sounds Step 1-esque, but it's asked directly.

[@manyasfon]

we can disregard the effect of furosemide on FeNa? because he is decompensated?

[@double_cheese_burger572]

love your outfit!💙

[@BuxAhmed]

Learning a lot from the videos.
I was wondering does long term pre-renal azotemia cause intra renal Azo?( I chose c based of that. )

[@bronxbomma718]

One of the tougher concepts I have had a problem with.....HAD. HAD. You made it go away!
GOAT.

[@jemilatBraimah]

Thank you Dr.💖

[@mohamedolama6037]

Thought na isn’t high due to lisinopril supressing raas system!

[@AngelaZavaro]

Fireeeee

[@whereisthemedicine]

I love your outfit 🫶

[@anusreedas4947]

Awesome

[@harismahmood4162]

Thanks

[@JudsonPaul-kz1oc]

ACE