Acute Phase Reactants (APRs) ESR and CRP
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- Опубліковано 20 лип 2024
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The serum level of acute phase reactants increase in inflammation and decrease when there is no inflammation.
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Serum half-life of CRP is 18 hours(Page 72,Davidson's 23rd edition)
Dr
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this video helped me figure out so many things that confused me for a long time about lab test results. Absolutely amazing.
Glad it was helpful! Thank you!
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Your explanation!! Like Narrating a story. "Every thing is for a reason"
I'm blessed☺ so that I found this video. Thanks a lot.
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Really wonderful ……….This video has two plus points i.e. short and fully conceptual video………..No rote learning……….Thanks sir for amazing explanation 🙏🙏
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Thanks for the great videos. I use these vids to study for my medical lab tests.
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These are excellent videos!! Maybe one suggestion using the color coding more purposefully and including more pictures. Otherwise these are very high yield vides thank you! :)
Hello , it is possible eventually to calculate in mVolt the value of Zeta potential in the blood ? Would be helpful some more blood test, also to understand general low grade inflammation ?
I'm studying the bioelectrical basis of health
One thing I didn’t understand that why you showed ceruloplasmin, haptoglobin & alfa 1 antitrypsin with positive APRs although they are decreased in acute phase. Please somebody explain this.
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God bless you too!
Hi, why is it that CRP rises first than WBC in some cases? And then when wbc becomes elevated, CRP decreases?
just a quick question, should the formation of the local blood clot occur outside the blood vessel? because if the coagulation occurs inside the blood vessel it can lead to thrombosis
such a wonderful video !😍
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Wilson is non inflammatory then how it affects cerruloplasmin?
Why in temporal vasculitis why we measure esr not crp
Excellent video! Thanks for doing It.
Ceruloplasmin levels are low in Wilson's :)
Thanks for your encouragement!
Regarding your answer: tell me why?
@@MedicosisPerfectionalis Well, its an hereditary disorder (cant Remember the gene) in which Ceruloplasmin can not be synthesize by the liver. Therefore, its level is drastically low and copper is really high, since is its carrier. :)
You’re excellent 👏
@@Unmercey1360 ATP7B gene
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I'm surprised you didn't mention prealbumin, another negative acute phase reactant. I didn't know antithrombin was, though, so thanks for that.
do you have video about ASTO?
Amazing way of teaching 🌟💯😇😇please make videos of all the topics sir 😇
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Is it possible that platelets also increase in acute inflammatory state, and thus can indrectly act as acute phase reactant ?
Platelet can rise in acute inflammation, it’s called secondary thrombocytosis.
@@MedicosisPerfectionalis thanks for reply 👍
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"Anti-thrombin, go to hell, we don't need you right now!" 😂😂😂
:)
excellent !!!
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عظمة!
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very perfect,,
Thanks 🙏
Thanks alot
Absolutely 👍
Ceruloplasmin is low in Wilson disease.
Elaine LUTHER Excellent!
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decrease ceruloplasmin in wilson
Excellent!
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It's the Nasty,ugly bacteria for me😂. Well explained 👏
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Ceruloplasmin level decrease in Wilson disease
Excellent!
hahahah....u r funny....i enjoyed watching it
Ansh Parabtani Thank you so much 😊
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3:18
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Excellent 👌 thanks a lot
My pleasure 😇
what is the answer?
CRP normalizes first.
@@MedicosisPerfectionalis ok then I wonder why I have high CRP but normal ESR
Watch at 1.25x
2x tbh
Low
CRP should absolutely replace ESR. It's an outdated test!!
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But CRP and ESR are used together since CRP can be normal in SLE,Multiple Myeloma,Sjogren's,UC,Pregnancy,End Stage Renal Disease but ESR is still high in these scenarios.
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